Arnica tea: a skin aid Health Dictionary

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Arnica Tea: A Skin Aid: From 1 Different Sources

Arnica tea is a healing beverage, with a long history in treating a large array of ailments. It is used only externally, because of its toxicity. Arnica Tea description Arnica is a woodland plant from the same family as the sunflower. It normally grows in Central Europe and in the western United States, at high altitudes. It is known as mountain tobacco or smoke herb, because Native American Indians used to smoke this herb. Arnica tea has been used since the 16^th century to calm digestive disorders, reduce fever and for topical treatment when dealing with skin disorders. At present, Arnica tea is recommended to be used only externally, because of its potential toxicity. Its flowers are dried and used to prepare ointments, creams and gels to treat bruises and sprains. Arnica Tea brewing To prepare Arnica tea:

Infuse 1 teaspoon of dried (or powdered) flower into ½ cup of hot water.
Allow the mix to steep for about 10 minutes.

Arnica tea should not be ingested or drunk. Its usage is only external. Arnica Tea benefits Arnica Tea is recognized for its anti-inflammatory and wound-healing properties. This tea has been successfully used to treat:

muscle pains
contusions
sore feet
leg ulcers in diabetics
sprains and bruises
hair loss
acne
scars, eczemas and itching caused by poison ivy

Arnica Tea side effects It has been proved that Arnica tea applied on open wounds or broken skin can increase the blood pressure. Arnica tea should be avoided in case of pregnancy. Also, people with sensitive skin are recommended not to use it. Long periods of Arnica tea usage can lead to eczema, edemas, rash, swelling and dermatitis. Arnica tea is a healing beverage which can heal skin problems and also, lessen pain. It is recommended to people looking for a medicinal remedy for their health issues.

Health Source: Beneficial Teas
Author: Health Dictionary

Aids

See Acquired Immune Deficiency Syndrome.... aids

Aids-related Complex

A variety of chronic symptoms and physical findings that occur in some persons who are infected with HIV, but do not meet the Centres for Disease Control’s definition of AIDS. Symptoms may include chronic swollen glands, recurrent fevers, unintentional weight loss, chronic diarrhoea, lethargy, minor alterations of the immune system (less severe than those that occur in AIDS), and oral thrush. ARC may or may not develop into AIDS.... aids-related complex

Hearing Aids

Nearly two-thirds of people aged over 70 have some degree of hearing impairment (see DEAFNESS). Hearing aids are no substitute for de?nitive treatment of the underlying cause of poor hearing, so examination by an ear, nose and throat surgeon and an audiologist is sensible before a hearing aid is issued (and is essential before one can be given through the NHS). The choice of aid depends on the age, manipulative skills, and degree of hearing impairment of the patient and the underlying cause of the deafness. The choice of hearing aid for a deaf child is particularly important, as impaired hearing can hinder speech development.

Electronic aids consist, essentially, of a microphone, an ampli?er, and an earphone. In postaural aids the microphone and ampli?er are contained in a small box worn behind the ear or attached to spectacles. The earphone is on a specially moulded earpiece. Some patients ?nd it di?cult to manipulate the controls of an aid worn behind the ear, and they may be better o? with a device worn on the body. Some hearing aids are worn entirely within the ear and are very discreet. They are particularly useful for people who have to wear protective headgear such as helmets.

The most sophisticated aids sit entirely within the ear canal so are virtually invisible. They may be tuned so that only the frequencies the wearer cannot hear are ampli?ed.

Many have a volume control and a special setting for use with telephone and in rooms ?tted with an inductive coupler that screens out background noise.

In making a choice therefore from the large range of e?ective hearing aids now available, the expert advice of an ear specialist must be obtained. The RNID (Royal National Institute for Deaf People) provides a list of clinics where such a specialist can be consulted. It also gives reliable advice concerning the purchase and use of hearing aids – a worthwhile function, as some aids are very expensive.... hearing aids

Skin

The membrane which envelops the outer surface of the body, meeting at the body’s various ori?ces, with the mucous membrane lining the internal cavities.

Structure

CORIUM The foundation layer. It overlies the subcutaneous fat and varies in thickness from 0·5–3.0 mm. Many nerves run through the corium: these have key roles in the sensations of touch, pain and temperature (see NEURON(E)). Blood vessels nourish the skin and are primarily responsible for regulating the body temperature. Hairs are bedded in the corium, piercing the epidermis (see below) to cover the skin in varying amounts in di?erent parts of the body. The sweat glands are also in the corium and their ducts lead to the surface. The ?brous tissue of the corium comprises interlocking white ?brous elastic bundles. The corium contains many folds, especially over joints and on the palms of hands and soles of feet with the epidermis following the contours. These are permanent throughout life and provide unique ?ngerprinting identi?cation. HAIR Each one has a root and shaft, and its varying tone originates from pigment scattered throughout it. Bundles of smooth muscle (arrectores pilorum) are attached to the root and on contraction cause the hair to stand vertical. GLANDS These occur in great numbers in the skin. SEBACEOUS GLANDS secrete a fatty substance and sweat glands a clear watery ?uid (see PERSPIRATION). The former are made up of a bunch of small sacs producing fatty material that reaches the surface via the hair follicle. Around three million sweat or sudoriparous glands occur all over the body surface; sited below the sebaceous glands they are unconnected to the hairs. EPIDERMIS This forms the outer layer of skin and is the cellular layer covering the body surface: it has no blood vessels and its thickness varies from 1 mm on the palms and soles to 0·1 mm on the face. Its outer, impervious, horny layer comprises several thicknesses of ?at cells (pierced only by hairs and sweat-gland openings) that are constantly rubbed o? as small white scales; they are replaced by growing cells from below. The next, clear layer forms a type of membrane below which the granular stratum cells are changing from their origins as keratinocytes in the germinative zone, where ?ne sensory nerves also terminate. The basal layer of the germinative zone contains melanocytes which produce the pigment MELANIN, the cause of skin tanning.

Nail A modi?cation of skin, being analagous to the horny layer, but its cells are harder and more adherent. Under the horny nail is the nail bed, comprising the well-vascularised corium (see above) and the germinative zone. Growth occurs at the nail root at a rate of around 0·5 mm a week – a rate that increases in later years of life.

Skin functions By its ability to control sweating and open or close dermal blood vessels, the skin plays a crucial role in maintaining a constant body temperature. Its toughness protects the body from mechanical injury. The epidermis is a two-way barrier: it prevents the entry of noxious chemicals and microbes, and prevents the loss of body contents, especially water, electrolytes and proteins. It restricts electrical conductivity and to a limited extent protects against ultraviolet radiation.

The Langerhans’ cells in the epidermis are the outposts of the immune system (see IMMUNITY), just as the sensory nerves in the skin are the outposts of the nervous system. Skin has a social function in its ability to signal emotions such as fear or anger. Lastly it has a role in the synthesis of vitamin D.... skin

Arnica

Leopard’s Bane. Wolf’s Bane. Arnica Montana L. German: Wolferlei, Arnika. French: Arnica, Aronique. Spanish: Arnica. Italian: Arnica, Polmonaria di Montagna. Dried flowerheads.

Action: external use only.

Uses: Bruises and contusions where skin is unbroken. Severe bruising after surgical operation. Neuralgia, sprains, rheumatic joints, aches and pains after excessive use as in sports and gardening.

Combination, in general use: 1 part Tincture Arnica to 10 parts Witch Hazel water as a lotion. Contra- indications: broken or lacerated skin.

Preparations: Compress: handful flowerheads to 1 pint boiling water. Saturate handtowel or suitable material in mixture and apply.

Tincture. 1 handful (50g) flowerheads to 1 pint 70 per cent alcohol (say Vodka) in wide-necked bottle. Seal tight. Shake daily for 7 days. Filter. Use as a lotion or compress: 1 part tincture to 20 parts water. Weleda Lotion. First aid remedy to prevent bruise developing.

Nelson’s Arnica cream.

Ointment. Good for applying Arnica to parts of the body where tincture or lotion is unsuitable. 2oz flowers and 1oz leaves (shredded or powdered) in 16oz lard. Moisten with half its weight of distilled water. Heat together with the lard for 3-4 hours and strain. For wounds and varicose ulcers.

Wet Dressing. 2 tablespoons flowers to 2 litres boiling water. For muscular pain, stiffness and sprains. Tincture. Alternative dosage: a weak tincture can be used with good effect, acceptable internally: 5 drops tincture to 100ml water – 1 teaspoon hourly or two-hourly according to severity of the case.

Widely used in Homoeopathic Medicine.

First used by Swiss mountaineers who chewed the leaves to help prevent sore and aching limbs.

Note: Although no longer used internally in the UK, 5-10 drop doses of the tincture are still favoured by some European and American physicians for anginal pain and other acute heart conditions; (Hawthorn for chronic).

Pharmacy only sale. ... arnica

Skin Graft

A technique used to repair areas of lost or damaged skin that are too large to heal naturally, that are slow

to heal, or that would leave tethering or unsightly scars. A skin graft is often used in the treatment of burns or sometimes for nonhealing ulcers. A piece of healthy skin is detached from one part of the body and transferred to the affected area. New skin cells grow from the graft and cover the damaged area. In a meshed graft, donor skin is removed and made into a mesh by cutting. The mesh is stretched to fit the recipient site; new skin cells grow to fill the spaces in the mesh. In a pinch graft, multiple small areas of skin are pinched up and removed from the donor site. Placed on the recipient site, they gradually expand to form a new sheet of healthy skin. (See also skin flap.)... skin graft

Acquired Immune Deficiency Syndrome (aids)

A severe manifestation of infection with the Human immunodeficiency virus (HIV).... acquired immune deficiency syndrome (aids)

Aida

(English / French / Arabic) One who is wealthy; prosperous / one who is helpful / a returning visitor

Ayda, Aydah, Aidah, Aidee, Aidia, Aieeda, Aaida... aida

Aidan

(Gaelic) One who is fiery; little fire Aiden, Adeen, Aden, Aideen, Adan, Aithne, Aithnea, Ajthne, Aedan, Aeden... aidan

Aids/hiv

Acquired Immune De?ciency Syndrome (AIDS) is the clinical manifestation of infection with Human Immunode?ciency Virus (HIV). HIV belongs to the retroviruses, which in turn belong to the lentiviruses (characterised by slow onset of disease). There are two main HIV strains: HIV-1, by far the commonest; and HIV-2, which is prevalent in Western Africa (including Ivory Coast, Gambia, Mali, Nigeria and Sierra Leone). HIV attacks the human immune system (see IMMUNITY) so that the infected person becomes susceptible to opportunistic infections, such as TUBERCULOSIS, PNEUMONIA, DIARRHOEA, MENINGITIS and tumours such as KAPOSI’S SARCOMA. AIDS is thus the disease syndrome associated with advanced HIV infection.

Both HIV-1 and HIV-2 are predominantly sexually transmitted and both are associated with secondary opportunistic infections. However, HIV-2 seems to result in slower damage to the immune system. HIV-1 is known to mutate rapidly and has given rise to other subtypes.

HIV is thought to have occurred in humans in the 1950s, but whether or not it infected humans from another primate species is uncertain. It became widespread in the 1970s but its latency in causing symptoms meant that the epidemic was not noticed until the following decade. Although it is a sexually transmitted disease, it can also be transmitted by intravenous drug use (through sharing an infected needle), blood transfusions with infected blood (hence the importance of e?ective national blood-screening programmes), organ donation, and occupationally (see health-care workers, below). Babies born of HIV-positive mothers can be infected before or during birth, or through breast feeding.

Although HIV is most likely to occur in blood, semen or vaginal ?uid, it has been found in saliva and tears (but not sweat); however, there is no evidence that the virus can be transmitted from these two body ?uids. There is also no evidence that HIV can be transmitted by biting insects (such as mosquitoes). HIV does not survive well in the environment and is rapidly destroyed through drying.

Prevalence At the end of 2003 an estimated 42 million people globally were infected with HIV – up from 40 million two years earlier. About one-third of those with HIV/AIDS are aged 15–24 and most are unaware that they are carrying the virus. During 2003 it is estimated that 5 million adults and children worldwide were newly infected with HIV, and that 3 million adults and children died. In Africa in 2003,

3.4 million people were newly infected and 2.3 million died, with more than 28 million carrying the virus. HIV/AIDS was the leading cause of death in sub-Saharan Africa where over half of the infections were in women and 90 per cent of cases resulted from heterosexual sex. In some southern African countries, one in three pregnant women had HIV.

In Asia and the Paci?c there were 1.2 million new infections and 435,000 deaths. The area with the fastest-growing epidemic is Eastern Europe, especially the Russian Federation where in 2002 around a million people had HIV and there were an estimated 250,000 new infections, with intravenous drug use a key contributor to this ?gure. Seventy-?ve per cent of cases occurred in men, with male-to-male sexual transmission an important cause of infection, though heterosexual activity is a rising cause of infection.

At the end of 2002 the UK had an estimated 55,900 HIV-infected adults aged between 15 and 59. More than 3,600 individuals were newly diagnosed with the infection in 2000, the highest annual ?gure since the epidemic started

– in 1998 the ?gure was 2,817 and in 1999 just over 3,000 (Department of Health and Communicable Disease Surveillance Centre). The incidence of AIDS in the UK has declined sharply since the introduction of highly active antiretroviral therapy (HAART) and HIV-related deaths have also fallen: in 2002 there were 777 reported new AIDS cases and 395 deaths, compared with 1,769 and 1,719 respectively in 1995. (Sources: UNAIDS and WHO, AIDS Epidemic Update, December 2001; Public Health Laboratory Services AIDS and STD Centre Communicable Disease Surveillance and Scottish Centre for Infection and Environmental Health, Quarterly Surveillance Tables.)

Poverty is strongly linked to the spread of AIDS, for various reasons including lack of health education; lack of e?ective public-health awareness; women having little control over sexual behaviour and contraception; and, by comparison with the developed world, little or no access to antiretroviral drugs.

Pathogenesis The cellular target of HIV infection is a subset of white blood cells called T-lymphocytes (see LYMPHOCYTE) which carry the CD4 surface receptor. These so-called ‘helper T-cells’ are vital to the function of cell-mediated immunity. Infection of these cells leads to their destruction (HIV replicates at an enormous rate – 109) and over the course of several years the body is unable to generate suf?cient new cells to keep pace. This leads to progressive destruction of the body’s immune capabilities, evidenced clinically by the development of opportunistic infection and unusual tumours.

Monitoring of clinical progression It is possible to measure the number of viral particles present in the plasma. This gives an accurate guide to the likely progression rate, which will be slow in those individuals with fewer than 10,000 particles per ml of plasma but progressively more rapid above this ?gure. The main clinical monitoring of the immune system is through the numbers of CD4 lymphocytes in the blood. The normal count is around 850 cells per ml and, without treatment, eventual progression to AIDS is likely in those individuals whose CD4 count falls below 500 per ml. Opportunistic infections occur most frequently when the count falls below 200 per ml: most such infections are treatable, and death is only likely when the CD4 count falls below 50 cells per ml when infection is developed with organisms that are di?cult to treat because of their low intrinsic virulence.

Simple, cheap and highly accurate tests are available to detect HIV antibodies in the serum. These normally occur within three months of infection and remain the cornerstone of the diagnosis.

Clinical features Most infected individuals have a viral illness some three weeks after contact with HIV. The clinical features are often non-speci?c and remain undiagnosed but include a ?ne red rash, large lymph nodes, an in?uenza-like illness, cerebral involvement and sometimes the development of opportunistic infections. The antibody test may be negative at this stage but there are usually high levels of virus particles in the blood. The antibody test is virtually always positive within three months of infection. HIV infection is often subsequently asymptomatic for a period of ten years or more, although in most patients progressive immune destruction is occurring during this time and a variety of minor opportunistic infections such as HERPES ZOSTER or oral thrush (see CANDIDA) do occur. In addition, generalised LYMPHADENOPATHY is present in a third of patients and some suffer from severe malaise, weight loss, night sweats, mild fever, ANAEMIA or easy bruising due to THROMBOCYTOPENIA.

The presentation of opportunistic infection is highly variable but usually involves either the CENTRAL NERVOUS SYSTEM, the gastrointestinal tract or the LUNGS. Patients may present with a sudden onset of a neurological de?cit or EPILEPSY due to a sudden onset of a STROKE-like syndrome, or epilepsy due to a space-occupying lesion in the brain – most commonly TOXOPLASMOSIS. In late disease, HIV infection of the central nervous system itself may produce progressive memory loss, impaired concentration and mental slowness called AIDS DEMENTIA. A wide variety of opportunistic PROTOZOA or viruses produces DYSPHAGIA, DIARRHOEA and wasting. In the respiratory system the commonest opportunistic infection associated with AIDS, pneumonia, produces severe shortness of breath and sometimes CYANOSIS, usually with a striking lack of clinical signs in the chest.

In very late HIV infection, when the CD4 count has fallen below 50 cells per ml, infection with CYTOMEGALOVIRUS may produce progressive retinal necrosis (see EYE, DISORDERS OF) which will lead to blindness if untreated, as well as a variety of gastrointestinal symptoms. At this stage, infection with atypical mycobacteria is also common, producing severe anaemia, wasting and fevers. The commonest tumour associated with HIV is Kaposi’s sarcoma which produces purplish skin lesions. This and nonHodgkin’s lymphoma (see LYMPHOMA), which is a hundred times more frequent among HIV-positive individuals than in the general population, are likely to be associated with or caused by opportunistic viral infections.

Prevention There is, as yet, no vaccine to prevent HIV infection. Vaccine development has been hampered

by the large number of new HIV strains generated through frequent mutation and recombination.

because HIV can be transmitted as free virus and in infected cells.

because HIV infects helper T-cells – the very cells involved in the immune response. There are, however, numerous research pro

grammes underway to develop vaccines that are either prophylactic or therapeutic. Vaccine-development strategies have included: recombinant-vector vaccines, in which a live bacterium or virus is genetically modi?ed to carry one or more of the HIV genes; subunit vaccines, consisting of small regions of the HIV genome designed to induce an immune response without infection; modi?ed live HIV, which has had its disease-promoting genes removed; and DNA vaccines – small loops of DNA (plasmids) containing viral genes – that make the host cells produce non-infectious viral proteins which, in turn, trigger an immune response and prime the immune system against future infection with real virus.

In the absence of an e?ective vaccine, preventing exposure remains the chief strategy in reducing the spread of HIV. Used properly, condoms are an extremely e?ective method of preventing exposure to HIV during sexual intercourse and remain the most important public-health approach to countering the further acceleration of the AIDS epidemic. The spermicide nonoxynol-9, which is often included with condoms, is known to kill HIV in vitro; however, its e?ectiveness in preventing HIV infection during intercourse is not known.

Public-health strategies must be focused on avoiding high-risk behaviour and, particularly in developing countries, empowering women to have more control over their lives, both economically and socially. In many of the poorer regions of the world, women are economically dependent on men and refusing sex, or insisting on condom use, even when they know their partners are HIV positive, is not a straightforward option. Poverty also forces many women into the sex industry where they are at greater risk of infection.

Cultural problems in gaining acceptance for universal condom-use by men in some developing countries suggests that other preventive strategies should also be considered. Microbicides used as vaginal sprays or ‘chemical condoms’ have the potential to give women more direct control over their exposure risk, and research is underway to develop suitable products.

Epidemiological studies suggest that male circumcision may o?er some protection against HIV infection, although more research is needed before this can be an established public-health strategy. Globally, about 70 per cent of infected men have acquired the virus through unprotected vaginal sex; in these men, infection is likely to have occurred through the penis with the mucosal epithelia of the inner surface of the foreskin and the frenulum considered the most likely sites for infection. It is suggested that in circumcised men, the glans may become keratinised and thus less likely to facilitate infection. Circumcision may also reduce the risk of lesions caused by other sexually transmitted disease.

Treatment AIDS/HIV treatment can be categorised as speci?c therapies for the individual opportunistic infections – which ultimately cause death – and highly active antiretroviral therapy (HAART) designed to reduce viral load and replication. HAART is also the most e?ective way of preventing opportunistic infections, and has had a signi?cant impact in delaying the onset of AIDS in HIV-positive individuals in developed countries.

Four classes of drugs are currently in use. Nucleoside analogues, including ZIDOVUDINE and DIDANOSINE, interfere with the activity of the unique enzyme of the retrovirus reverse transcriptase which is essential for replication. Nucleotide analogues, such as tenofovir, act in the same way but require no intracellular activation. Non-nucleoside reverse transcriptase inhibitors, such as nevirapine and EFAVIRENZ, act by a di?erent mechanism on the same enzyme. The most potent single agents against HIV are the protease inhibitors, such as lopinavir, which render a unique viral enzyme ineffective. These drugs are used in a variety of combinations in an attempt to reduce the plasma HIV viral load to below detectable limits, which is achieved in approximately 90 per cent of patients who have not previously received therapy. This usually also produces a profound rise in CD4 count. It is likely, however, that such treatments need to be lifelong – and since they are associated with toxicities, long-term adherence is di?cult. Thus the optimum time for treatment intervention remains controversial, with some clinicians believing that this should be governed by the viral load rising above 10,000 copies, and others that it should primarily be designed to prevent the development of opportunistic infections – thus, that initiation of therapy should be guided more by the CD4 count.

It should be noted that the drug regimens have been devised for infection with HIV-1; it is not known how e?ective they are at treating infection with HIV-2.

HIV and pregnancy An HIV-positive woman can transmit the virus to her fetus, with the risk of infection being particularly high during parturition; however, the risk of perinatal HIV transmission can be reduced by antiviral drug therapy. In the UK, HIV testing is available to all women as part of antenatal care. The bene?ts of antenatal HIV testing in countries where antiviral drugs are not available are questionable. An HIV-positive woman might be advised not to breast feed because of the risks of transmitting HIV via breastmilk, but there may be a greater risk associated with not breast feeding at all. Babies in many poor communities are thought to be at high risk of infectious diseases and malnutrition if they are not breast fed and may thus be at greater overall risk of death during infancy.

Counselling Con?dential counselling is an essential part of AIDS management, both in terms of supporting the psychological wellbeing of the individual and in dealing with issues such as family relations, sexual partners and implications for employment (e.g. for health-care workers). Counsellors must be particularly sensitive to culture and lifestyle issues. Counselling is essential both before an HIV test is taken and when the results are revealed.

Health-care workers Health-care workers may be at risk of occupational exposure to HIV, either through undertaking invasive procedures or through accidental exposure to infected blood from a contaminated needle (needlestick injury). Needlestick injuries are frequent in health care – as many as 600,000 to 800,000 are thought to occur annually in the United States. Transmission is much more likely where the worker has been exposed to HIV through a needlestick injury or deep cut with a contaminated instrument than through exposure of mucous membranes to contaminated blood or body ?uids. However, even where exposure occurs through a needlestick injury, the risk of seroconversion is much lower than with a similar exposure to hepatitis C or hepatitis B. A percutaneous exposure to HIV-infected blood in a health-care setting is thought to carry a risk of about one infection per 300 injuries (one in 1,000 for mucous-membrane exposure), compared with one in 30 for hepatitis C, and one in three for hepatitis B (when the source patient is e-antigen positive).

In the event of an injury, health-care workers are advised to report the incident immediately where, depending on a risk assessment, they may be o?ered post-exposure prophylaxis (PEP). They should also wash the contaminated area with soap and water (but without scrubbing) and, if appropriate, encourage bleeding at the site of injury. PEP, using a combination of antiretroviral drugs (in a similar regimen to HAART – see above), is thought to greatly reduce the chances of seroconversion; it should be commenced as soon as possible, preferably within one or two hours of the injury. Although PEP is available, safe systems of work are considered to o?er the greatest protection. Double-gloving (latex gloves remove much of the blood from the surface of the needle during a needlestick), correct use of sharps containers (for used needles and instruments), avoiding the resheathing of used needles, reduction in the number of blood samples taken from a patient, safer-needle devices (such as needles that self-blunt after use) and needleless drug administration are all thought to reduce the risk of exposure to HIV and other blood-borne viruses. Although there have been numerous cases of health-care workers developing HIV through occupational exposure, there is little evidence of health-care workers passing HIV to their patients through normal medical procedures.... aids/hiv

Cutaneous Means Belonging To The Skin.

... cutaneous means belonging to the skin.

Home Health Aide

A person who, under the supervision of a home health or social service agency, assists an older, ill or disabled person with household chores, bathing, personal care and other daily living needs. See also “community-based service”.... home health aide

Nurse Assistant / Aide

A staff member who has completed a specific requirement of coursework and clinical training and is responsible for lower levels of nursing care and assisting individuals with their daily living activities, such as bathing, toileting, eating and moving about.... nurse assistant / aide

Scalded-skin Syndrome

In infants, certain staphylococcal bacteria (see STAPHYLOCOCCUS) can cause an acute toxic illness in which the subject develops sheets of bright ERYTHEMA, accompanied by shedding of layers of outer epidermis. The result is similar to a hot-water scald. The condition responds promptly to appropriate antibiotic therapy. Drug reactions, especially from sulphonamides, may cause a similar syndrome in adults. In drug-induced forms, mucosae are also affected and the disease is often fatal.... scalded-skin syndrome

Greasy Skin

Blue Flag, Goldenseal, Queen’s Delight, Garlic. ... greasy skin

Callus, Skin

An area of thickened skin, usually on the hands or feet, caused by regular or prolonged pressure or friction.

A corn is a callus on a toe.

If corns are painful, the thickened skin can be pared away by a chiropodist using a scalpel.... callus, skin

Chapped Skin

Sore, cracked, rough skin, usually on the hands, face, and particularly the lips, due to dryness. Chapping is caused by the lack, or removal, of the natural oils that keep skin supple. It tends to occur in cold weather, when oil-secreting glands produce less oil, or after repeated washing or wetting. Treatment is with a lanolin-based cream.... chapped skin

Skin Allergy

Irritation of the skin following contact with a specific substance that provokes an inappropriate or exaggerated reaction from the immune system. There are 2 main types of allergic skin reaction. In contact allergic dermatitis, red, itchy patches develop a few hours to 2 days after contact with the allergen. In contact urticaria, red, raised areas appear a few minutes after skin contact. In some cases, skin tests are needed, to identify the allergen, for contact with it to be minimized. (See also atopic eczema.)... skin allergy

Skin Biopsy

Removal of a portion of skin for laboratory analysis in order to diagnose a skin disorder.... skin biopsy

Skin Cancer

A malignant tumour in the skin. Basal cell carcinoma, squamous cell carcinoma, and malignant melanoma are common forms related to long-term exposure to sunlight. Bowen’s disease, a rare disorder that can become cancerous, may also be related to sun exposure. Less common types include Paget’s disease of the nipple and mycosis fungoides. Kaposi’s sarcoma is a type usually found in people with AIDS. Most skin cancers can be cured if treated early.... skin cancer

Skin-grafting

An operation in which large breaches of SKIN surface due to wounding, burns or ulceration are closed by TRANSPLANTATION of skin from other parts. There are three methods by which this is done. Most frequently the epidermis only is transplanted, using a method introduced by Reverdin and by Thiersch, and known by their names. For this purpose, a broad strip of epidermis is shaved o? the thigh or upper arm, after the part has been carefully sterilised, and is transferred bodily to the raw or ulcerated surface, or is cut into smaller strips and laid upon it. A second method is for small pieces of the skin in its whole thickness to be removed from the arm and thigh, or even from other people, and then implanted and bound upon the raw surface. (This method has the disadvantage that the true skin must contract at the spot from which the graft is taken, leaving an unsightly scar.) When very large areas require to be covered, a third method is commonly used. A large ?ap of skin, amply su?cient to cover the gap, is raised from a neighbouring or distant part of the body, in such a way that it remains attached along one margin, so that blood vessels can still enter and nourish it. It is then turned so as to cover the gap; or, if it be situated on a distant part, the two parts are brought together and ?xed in this position until the ?ap grows ?rmly to its new bed. The old connection of the ?ap is then severed, leaving it growing in its new place.

Researchers are having success in growing human skin in the laboratory for grafting on to people who have been badly burned and have insu?cient intact skin surface to provide an autologous graft (one provided by the recipient of the graft). Other techniques being researched are the use of specially treated shark skin and the production of arti?cial skin.... skin-grafting

Skin, Disorders Of The

The skin is vulnerable to various disorders, including birthmarks and other naevi; infections that may be viral (such as cold sores and warts), bacterial (for example, cellulitis), or fungal (such as tinea, which causes athlete’s foot); rashes due to vitamin deficiency or the side effects of drugs; and tumours, both noncancerous and cancerous.

Acne is common in adolescents and is partly related to the action of androgen hormones.

Inflammation of the skin occurs in dermatitis, eczema, and skin allergy.

The skin is also vulnerable to injuries such as burns, cuts, and bites (see bites, animal; insect bites).... skin, disorders of the

Skin Flap

A surgical technique in which a section of skin and underlying tissue, sometimes including muscle, is moved to cover an area from which skin and tissue have been lost or damaged by injury, disease, or surgery.

Unlike a skin graft, a skin flap retains its blood supply, either by remaining attached to the donor site or through reattachment to blood vessels at the recipient site by microsurgery, so skin flaps adhere well even where there is extensive loss of deep tissue.... skin flap

Skin Patch

See transdermal patch.... skin patch

Skin Peeling, Chemical

A cosmetic operation in which the outer layers of the skin are peeled away by the application of a caustic paste in order to remove freckles, acne scars, delicate wrinkles, or other skin blemishes.... skin peeling, chemical

Skin, Diseases Of

They may be local to the SKIN, or a manifestation of systemic disorders – inherited or acquired. Some major types are described below.

Others appear under their appropriate alphabetical headings: ACNE; ALBINISM; ALOPECIA; ALOPECIA AREATA; APHTHOUS ULCER; BASAL CELL CARCINOMA; BOILS (FURUNCULOSIS); BOWEN’S DISEASE; CALLOSITIES; CANDIDA; CHEILOSIS; CHEIRAPOMPHOLYX; DANDRUFF; DERMATOFIBROMA; DERMATOMYOSITIS; DERMATOPHYTES; DERMOGRAPHISM; ECTHYMA; ERYSIPELAS; ERYTHEMA; ERYTHRASMA; ERYTHRODERMA; ESCHAR; EXANTHEM; FUNGAL AND YEAST INFECTIONS; HAND, FOOT AND MOUTH DISEASE; HERPES GENITALIS; HERPES SIMPLEX; HERPES ZOSTER; IMPETIGO; INTERTRIGO; KELOID; KERATOSIS; LARVA MIGRANS; LICHEN; LUPUS; MADURA FOOT; MELANOMA; MILIARIA; MOLLUSCUM CONTAGIOSUM; MOLE; MYCOSIS FUNGOIDES; NAEVUS; ORF; PEDICULOSIS; PEMPHIGUS; PHOTOCHEMOTHERAPY; PHOTODERMATOSES; PITYRIASIS; PORPHYRIAS; PRURITUS; PSORIASIS; RINGWORM; ROSACEA; SARCOIDOSIS; SCABIES; SCLERODERMA; URTICARIA; VITILIGO; WARTS; XANTHOMATA.

Skin cancer Primary cancer is common and chronic exposure to ultraviolet light is the most important cause. BASAL CELL CARCINOMA is the most common form; squamous cell carcinoma is less common and presents as a growing, usually painless nodule which may ulcerate. Squamous cancer may spread to regional lymph glands and metastasise, unlike basal cell cancer. Occupational exposure to chemical carcinogens may cause squamous carcinoma – for example, cancer from pitch warts or the scrotal carcinoma of chimney sweeps exposed to coal dust in earlier centuries. Squamous carcinoma of the lip is associated with clay-pipe smoking.

Cancer may arise from the population of melanocytes of the skin (see MELANOCYTE; MELANOMA).

Apart from these three most frequent forms of skin cancer, various forms of cancer can arise from cells of the dermis, of which LYMPHOMA is the most important (see also MYCOSIS FUNGOIDES).

Lastly, secondary deposits from internal cancer, particularly from the breast, may metastasise to the skin.

Dermatitis and eczema These are broadly synonymous, and the terms are frequently interchangeable. Eczema is a pattern of in?ammation with many potential causes. Dermatitis is commonly used to suggest an eczema caused by external factors; it is a common pattern of in?ammation of the skin characterised by redness and swelling, vesiculation (see VESICLE), and scaling with intense itching and often exudation (weeping). Fissuring, thickening (licheni?cation – see LICHEN) and secondary bacterial infection may follow. Dermatitis can affect any part of the body. It may be genetically detemined or due to other ‘internal’ factors, such as venous HYPERTENSION in a leg, or stress. Often it is ‘external’ in origin – due to strong irritants or chemical allergens. (See also ALLERGY; ALLERGEN.) ATOPIC DERMATITIS is genetic in origin and usually begins in infancy. It may persist for years, and ASTHMA, allergic RHINITIS and conjunctivitis (see under EYE, DISORDERS OF) – ‘hay fever’ – may be associated. Atopic children tend to have multiple allergies, especially to inhaled allergens such as house-dust mite, cat and dog dander and pollens. Allergy to foods is less common but potentially more dangerous, especially if to nuts, when it can cause acute URTICARIA or even ANAPHYLAXIS. Atopic subjects are particularly prone to persistent and multiple verrucae (see WARTS) and mollusca (see MOLLUSCUM CONTAGIOSUM) and to severe HERPES SIMPLEX infections. (See also ATOPY.)

EXFOLIATE DERMATITIS (PITYRIASIS RUBRA)

Generalised exfoliation and scaling of the skin, commonly with ERYTHEMA. Drugs may cause it, or the disorder may be linked with other skin diseases such as benign dermatoses and lupus erythematosus (see under LUPUS). SUMMER POMPHOLYX is an acute vesicular eczema of the palms and soles recurring every summer. Inhaled allergens are a frequent cause. VENOUS (STASIS) DERMATITIS begins on a lower calf, often in association with PURPURA, swelling and sometimes ulceration. Chronic venous hypertension in the leg, consequent on valvular incompetence in the deep leg veins owing to previous deep vein thrombosis (see VEINS, DISEASES OF), is the usual cause. NEURODERMATITIS A pattern of well-de?ned plaques of licheni?ed eczema particularly seen on the neck, ulnar forearms or sides of the calves in subjects under emotional stress. IRRITANT CONTACT DERMATITIS Most often seen in an industrial setting (occupational dermatitis), it is due to damage by strong chemicals such as cutting oils, cement, detergents and solvents. In almost all cases the hands are most severely affected. ALLERGIC CONTACT DERMATITIS, in contrast, can affect any part of the body depending on the cause – for example, the face (cosmetics), hands (plants, occupational allergens) or soles (rubber boots). Particularly common allergens include metals (nickel and chromate), rubber addititives, and adhesives (epoxy resins).

Treatment Avoidance of irritants and contact allergens, liberal use of EMOLLIENTS, and topical application of corticosteroid creams and ointments (see CORTICOSTEROIDS) are central.... skin, diseases of

Skin Tag

A harmless, small, brown or flesh-coloured flap of skin that may appear spontaneously or as a result of poor healing of a wound.... skin tag

Skin Tests

Procedures for determining the body’s reaction to various substances by injecting a small quantity of the substance under the skin or by applying it to the skin (usually on patches).

Patch tests are used in the diagnosis of contact allergic dermatitis.

They can also be used to test immunity to certain infectious diseases (such as in the tuberculin test).... skin tests

Skin Tumours

A growth on or in the skin that may be cancerous (see skin cancer) or noncancerous.

Keratoses and squamous papillomas are common types of noncancerous tumour; other types include sebaceous cysts, cutaneous horns, keratoacanthomas, and haemangiomas.... skin tumours

Walking Aids

Equipment for increasing the mobility of people who have a disorder that affects their ability to walk. Aids include walking sticks, crutches, and walking frames.... walking aids

Split-skin Graft

(SSG, Thiersch’s graft) a type of skin graft in which thin partial thicknesses of skin are used to cover and heal a wound. They are removed from one site on the body, cut into narrow strips or sheets, and placed onto the wound area to be healed.... split-skin graft

Tea For Good Skin

Good skin is something we all want. Even if you’re a man or a woman, black heads or acne are really bothering you. If that is the case, you have definitely tried a lot of pharmaceutical and cosmetic products and nothing seemed to be working. Alternative medicine recommends a number of teas and decoctions that will make your skin smoother than ever. All you have to do is commit to this treatment and maintain an adequate skin hygiene. How Tea for Good Skin Works It’s important to know that not only acne can deteriorate your natural glow. There are also a number of affections that can stain your skin, such as liver or kidney failure, eczema or skin rash due to allergies. Smoke will age you before time, wrinkling your mouth area and your forehead. Also, you may want to change your pillow case more often, in order to keep allergens and microbes away from your face. A Tea for Good Skin’s main purpose is to clear your skin through its antiseptic ingredients and nourish the damaged areas. Efficient Tea for Good Skin When choosing a Tea for Good Skin, you need to pick the ones with the highest antifungal and antiseptic properties. You can either drink the tea or use it as a face cleanser. In case you don’t know which teas are adequate, here’s a list we made for you: - Chamomile Tea – thanks to its antibacterial and antiseptic properties, Chamomile Tea is a great help when it comes to skin treatments. Both the pharmaceutical and the cosmetic companies have included Chamomile on their must have list of ingredients. A cup of tea per day will restore your skin’s natural glow while also improving your general health. - Oolong Tea - contains half the amount of caffeine that other teas contain. You can drink it daily or use it as a compress to apply it on your affected areas. This is probably the most effective Tea for Good Skin and also the safest. If you haven’t tried it yet, now would be a good time! - Black Tea – this wonderful Tea for skin improves your vascular activity and enhances your epithelial cells production. Pay attention, though: don’t take it if you’re on your period or experiencing some menopausal pains in order to avoid complication! - White Tea – also a good nutrient, White Tea can improve your general health, not just your skin. It’s best not to combine it with other tea, though. White Tea can have a negative reaction when mixed with green tea or black tea. You can also use a decoction or White Tea tinctures in order to treat your localized injuries. Tea for Good Skin Side Effects When taken properly, these teas are perfectly safe. Just make sure you don’t exceed the number of cups recommended per day in order to avoid complications such as diarrhea or constipation. Other than that, there’s no reason not to try a face cleanser based on a Tea for Good skin! However, if you’re not sure about it yet, talk to a dermatologist or to an herbalist. Don’t take a Tea for Good Skin if you’re pregnant, breastfeeding, on blood thinners, anticoagulants or preparing for a major surgery. If there’s nothing that could interfere with your herbal treatment, choose a Tea for Good Skin that fits you best and enjoy its wonderful benefits!... tea for good skin

Cancer – Skin

There is strong evidence that sunlight plays a major role in the development of human skin cancers. Skin malignancy usually takes the form of Basal Cell carcinoma, squamous cell carcinoma and melanoma that may develop from pre-existing naevi.

Basal Cell Cancer. Strong sunlight on fair skins. Common on face and hands and other exposed areas. Commences as a tiny hard nodule. See – RODENT ULCER.

Squamous Cell Cancer. The role of sunlight in this type of cancer is even more positive. Other causes: photosensitisers such as pitch and PUVA photochemotherapy. Commences as a raised scaly rapidly- growing nodule.

Malignant Melanoma. Rare, but incidence rising. Four different kinds. Incidence is increased in individuals with fair or red hair who tend to burn rather than tan in the sun.

Causes may be numerous: genetic, occupational hazards or exposure to low-level radiation. Heavy freckling in youth doubles the risk. (Western Canada Melanoma study)

A study carried out by the New York’s Memorial-Sloan Kettering Cancer Centre refers to damage to the ultra violet-blocking ozone layer by supersonic jet exhaust and aerosol propellants that can also raise the malignant melanoma rate. A University of Sydney study links fluorescent lighting with the disease. Symptoms. Itching lesion increases in size and with growing discoloration. Colours may present as brown, black, red, blue, white, with a red inflammatory border. May progress to a dry crust, with bleeding.

Study. A study conducted by a team from Melbourne University, Anti-Cancer Council and St Vincent’s Hospital, Australia, describes a summer-long experiment that showed that people who used a sun-screen lotion (in this case SPF-17) cut their chances of developing the first signs of skin cancer.

Study. Patients who receive blood transfusions are more likely to develop malignant lymphomas and non- melanomatous skin cancers. (European Journal of Cancer (Nov 1993))

Eclectic physicians of the 19th century reported success from the use of American Mandrake (podophylum peltatum). Recent experience includes a 76 per cent cure rate achieved in 68 patients with carcinoma of the skin by treatment twice daily for 14 days with an ointment consisting of Podophyllum resin 20 per cent, and Linseed oil 20 per cent, in lanolin, followed by an antibiotic ointment. (Martindale 27; 1977, p. 1341) Podophyllum is an anti-mitotic and inhibits cell-division and should not be applied to normal cells.

Aloe Vera. Fresh cut leaf, or gel, to wipe over exposed surfaces.

Vitamin E oil. Applying the oil to the skin can reduce chances of acquiring skin cancer from the sun. (University of Arizona College of Medicine)

Red Clover. “I have seen a case of skin cancer healed by applying Red Clover blossoms. After straining a strong tea, the liquid was simmered until it was the consistency of tar. After several applications the skin cancer was gone, and has not returned.” (May Bethel, in “Herald of Health”, Dec. 1963)

Clivers. Equal parts juice of Clivers (from juice extractor) and glycerine. Internally and externally.

Thuja. Internal: 3-5 drops Liquid Extract, morning and evening.

Topical. “Take a small quantity powdered Slippery Elm and add Liquid Extract Thuja to make a stiff paste. Apply paste to the lesion. Cover with gauze and protective covering. When dry remove pack and follow with compresses saturated with Thuja.” (Ellingwood’s Therapeutist, Vol 10, No 6, p. 212) Echinacea and Thuja. Equal parts liquid extracts assist healthy granulation and neutralise odour.

Rue Ointment. Simmer whole fresh leaves in Vaseline.

Poke Root. An old physician laid great stress on the use of concentrated juice of green leaves. Leaves are bruised, juice extracted, and concentrated by slow evaporation until the consistency of a paste, for persistent skin cancer. Care should be taken to confine to the distressed area. (Ellingwood’s Therapeutist, Vol 8, No 7, p. 275)

Maria Treben. Horsetail poultice.

Laetrile. Some improvement claimed. 1 gram daily.

Cider vinegar. Anecdotal evidence: external use: small melanoma.

Diet. See: DIET – CANCER. Beta-carotene foods.

Treatment by skin specialist or oncologist. ... cancer – skin

Candida, Of Skin And Nails

Infection by Candida albicans.

Internal. Goldenseal 1; Myrrh 1; Thuja half; Poke root half. Dose – Powders: 500mg (two 00 capsules or one-third teaspoon). Liquid extracts: one 5ml teaspoon. Tinctures: two 5ml teaspoons. Thrice daily before meals.

Capricin. See entry.

Topical. Thuja lotion: 1 teaspoon Liquid extract Thuja to 1oz (30ml) distilled extract Witch Hazel.

Aloe Vera; fresh juice or gel.

Tea Tree oil; may be diluted many times.

Comfrey cream; Castor oil, Oil of Mullen or Houseleek. Cider vinegar.

Night wash. Warm water to which is added a few drops Tincture Myrrh, Tincture Thuja or Tea Tree oil. Diet and Supplements. Same as for CANDIDA – VAGINAL. ... candida, of skin and nails

Diet - Skin Disorders

Low fat, low salt, high fibre. Dairy-free (no milk, cream, cheese, eggs). Soya milk is more suitable for children and adults than cow’s milk and provides protein, calories, calcium and vitamins. Polyunsaturates: oils of safflower, corn, Soya, sunflower seed, etc which are rich in essential fatty acids, low levels of which are frequently found in the blood of those with chronic skin disorders. Evening Primrose oil is a rich source of EFAs. Gluten-free diet has proved successful in some cases.

Accept. Goat’s milk, yoghurt, eggs – twice weekly. The high potassium and low salt content of bananas help reduce itching. Lecithin. Oily fish. Purslane is a non-fish source of EPA and suitable for the vegetarian approach. Cottage cheese. Pumpkin seeds as a source of zinc. Dandelion coffee. Artichoke: such as Schoenenberger plant juice. Salad dressing: emulsify 1 teaspoon Cider vinegar to each 2 teaspoons safflower seed oil.

Reject: Fried and greasy foods, pastries, chocolates, sweets, ice cream, spicy foods, seasoning, sausage meats, white flour products, white sugar products, alcoholic drinks, meat from the pig (ham, pork, bacon), peppers, horseradish, condiments. Powdered kelp in place of salt, powdered garlic or celery.

Foods known to contain artificial colours and preservatives. All soft drinks, except those made at home from fresh fruits or raw vegetables; coffee, strong tea, oranges. Cola drinks, chocolate, milk, cream, cheese, whey.

Supplement. Beta carotene.

Study. A flare-up can be caused by nuts, jams, fruits, artificially coloured or flavoured foods. (British Journal of Dermatology, 110, 457, (1984)) ... diet - skin disorders

Computer-aided Diagnosis

The use of computer technology in diagnostic tests and procedures.

Probability-based computer systems store information on thousands of cases of different disorders detailing exact type, location, duration, symptoms, medical history, and diagnosis.

A patient’s symptoms and medical history can be entered into a computer, which then compares the details with existing data and produces a list of the most likely diagnoses.

Such technology is not currently in common use in hospitals, but is of value for people isolated from medical services, such as oil-rig crews.

Computers programmed to interpret visual data, such as abnormal cells, have potential use in certain types of blood test and cervical smear tests.

Computers are also used in investigative procedures such as CT scanning and MRI.... computer-aided diagnosis

Staphylococcal Scalded Skin Syndrome

(Ritter’s disease) a potentially serious condition of young infants (and occasionally seen in adults) in which the skin becomes reddened and tender and then peels off, giving the appearance of a scald. The area of skin loss may be quite extensive and is usually centred on the armpits and groin. The underlying cause is an infection by certain bacteria of the genus *Staphylococcus. It is contagious and may occur in clusters. Treatment is by antibiotics (usually intravenous), but careful nursing is essential to prevent skin damage. Admission to hospital is mandatory for small children.... staphylococcal scalded skin syndrome

Skin Care

Acne (M,S,F,B,I,N):

Bergamot, camphor (white), cananga, cedarwood (Atlas, Texas & Virginian), chamomile (German & Roman), clove bud, galbanum, geranium, grapefruit, immortelle, juniper, lavandin, lavender (spike & true), lemon, lemongrass, lime, linaloe, litsea cubeba, mandarin, mint (peppermint & spearmint), myrtle, niaouli, palmarosa, patchouli, petitgrain, rosemary, rosewood, sage (clary & Spanish), sandalwood, tea tree, thyme, vetiver, violet, yarrow, ylang ylang.

Allergies (M,S,F,B,I):

Melissa, chamomile (German & Roman), immortelle, true lavender, spikenard.

Athlete’s foot (S):

Clove bud, eucalyptus, lavender (true &spike), lemon, lemongrass, myrrh, patchouli, tea tree.

Baldness & hair care (S,H):

West Indian bay, white birch, cedarwood (Atlas, Texas & Virginian), chamomile (German & Roman), grapefruit, juniper, patchouli, rosemary, sage (clary & Spanish), yarrow, ylang ylang.

Boils, abscesses & blisters (S,C,B):

Bergamot, chamomile (German & Roman), eucalyptus blue gum, galbanum, immortelle, lavandin, lavender (spike & true), lemon, mastic, niaouli, clary sage, tea tree, thyme, turpentine.

Bruises (S,C):

Arnica (cream), borneol, clove bud, fennel, geranium, hyssop, sweet marjoram, lavender, thyme.

Burns (C,N):

Canadian balsam, chamomile (German & Roman), clove bud, eucalyptus blue gum, geranium, immortelle, lavandin, lavender (spike & true), marigold, niaouli, tea tree, yarrow.

Chapped & cracked skin (S,F,B):

Peru balsam, Tofu balsam, benzoin, myrrh, patchouli, sandalwood.

Chilblains (S,N):

Chamomile (German & Roman), lemon, lime, sweet marjoram, black pepper.

Cold sores/herpes (S):

Bergamot, eucalyptus blue gum, lemon, tea tree.

Congested & dull skin (M,S,F,B,I):

Angelica, white birch, sweet fennel, geranium, grapefruit, lavandin, lavender (spike & true), lemon, lime, mandarin, mint (peppermint & spearmint), myrtle, niaouli, orange (bitter & sweet), palmarosa, rose (cabbage & damask), rosemary, rosewood, ylang ylang.

Cuts/sores (S,C):

Canadian balsam, benzoin, borneol, cabreuva, cade, chamomile (German & Roman), clove bud, elemi, eucalyptus (blue gum, lemon & peppermint), galbanum, geranium, hyssop, immortelle, lavender (spike & true), lavandin, lemon, lime, linaloe, marigold, mastic, myrrh, niaouli, Scotch pine, Spanish sage, Levant styrax, tea tree, thyme, turpentine, vetiver, yarrow.

Dandruff (S,H):

West Indian bay, cade, cedarwood (Atlas, Texas & Virginian), eucalyptus, spike lavender, lemon, patchouli, rosemary, sage (clary & Spanish), tea tree.

Dermatitis (M,S,C,F,B):

White birch, cade, cananga, carrot seed, cedarwood (Atlas, Texas & Virginian), chamomile (German & Roman), geranium, immortelle, hops, hyssop, juniper, true lavender, linaloe, litsea cubeba, mint (peppermint & spearmint), palmarosa, patchouli, rosemary, sage (clary & Spanish), thyme.

Dry & sensitive skin (M,S,F,B):

Peru balsam, Tolu balsam, cassie, chamomile (German & Roman), frankincense, jasmine, lavandin, lavender (spike & true), rosewood, sandalwood, violet.

Eczema (M,S,F,B):

Melissa, Peru balsam, Tolu balsam, bergamot, white birch, cade, carrot seed, cedarwood (Atlas, Texas & Virginian), chamomile (German & Roman), geranium, immortelle, hyssop, juniper, lavandin, lavender (spike & true), marigold, myrrh, patchouli, rose (cabbage & damask), rosemary, Spanish sage, thyme, violet, yarrow.

Excessive perspiration (S,B):

Citronella, cypress, lemongrass, litsea cubeba, petitgrain, Scotch pine, Spanish sage.

Greasy or oily skin/scalp (M,S,H,F,B):

West Indian bay, bergamot, cajeput, camphor (white), cananga, carrot seed, citronella, cypress, sweet fennel, geranium, jasmine, juniper, lavender, lemon, lemongrass, !itsea cubeba, mandarin, marigold, mimosa, myrtle, niaouli, palmarosa, patchouli, petitgrain, rosemary, rosewood, sandalwood, clary sage, tea tree, thyme, vetiver, ylang ylang.

Haemorrhoids/piles (S,C,B):

Canadian balsam, Copaiba balsam, coriander, cubebs, cypress, geranium, juniper, myrrh, myrtle, parsley, yarrow.

Insect bites (S,N):

French basil, bergamot, cajeput, cananga, chamomile (German & Roman), cinnamon leaf, eucalyptus blue gum, lavandin, lavender (spike & true), lemon, marigold, melissa, niaouli, tea tree, thyme, ylang ylang.

Insect repellent (S,V):

French basil, bergamot, borneol, camphor (white), Virginian cedarwood, citronella, clove bud, cypress, eucalyptus (blue gum & lemon), geranium, lavender, lemongrass, litsea cubeba, mastic, melissa, patchouli, rosemary, turpentine.

Irritated & inflamed skin (S,C,F,B):

Angelica, benzoin, camphor (white), Atlas cedarwood, chamomile (German & Roman), elemi, immortelle, hyssop, jasmine, lavandin, true lavender, marigold, myrrh, patchouli, rose (cabbage & damask), clary sage, spikenard, tea tree, yarrow.

Lice (S,H):

Cinnamon leaf, eucalyptus blue gum, galbanum, geranium, lavandin, spike lavender, parsley, Scotch pine, rosemary, thyme, turpentine.

Mouth & gum infections/ulcers (S,C):

Bergamot, cinnamon leaf, cypress, sweet fennel, lemon, mastic, myrrh, orange (bitter & sweet), sage (clary & Spanish), thyme.

Psoriasis (M,S,F,B):

Angelica, bergamot, white birch, carrot seed, chamomile (German & Roman), true lavender.

Rashes (M,S,C,F,B):

Peru balsam, Tofu balsam, carrot seed, chamomile (German & Roman), hops, true lavender, marigold, sandalwood, spikenard, tea tree, yarrow.

Ringworm (S,H):

Geranium, spike lavender, mastic, mint (peppermint & spearmint), myrrh, Levant styrax, tea tree, turpentine.

Scabies (S):

Tolu balsam, bergamot, cinnamon leaf, lavandin, lavender (spike & true), lemongrass, mastic, mint (peppermint & spearmint), Scotch pine, rosemary, Levant styrax, thyme, turpentine.

Scars & stretch marks (M,S):

Cabreuva, elemi, frankincense, galbanum, true lavender, mandarin, neroli, palmarosa, patchouli, rosewood, sandalwood, spikenard, violet, arrow.

Slack tissue (M,S,B):

Geranium, grapefruit, juniper, lemongrass, lime, mandarin, sweet marjoram, orange blossom, black pepper, petitgrain, rosemary, yarrow.

Spots (S,N):

Bergamot, cade, cajeput, camphor (white), eucalyptus (lemon), immortelle, lavandin, lavender (spike & true), lemon, lime, litsea cubeba, mandarin, niaouli, tea tree.

Ticks (S,N):

Sweet marjoram.

Toothache & teething pain (S,C,N):

Chamomile (German & Roman), clove bud, mastic, mint (peppermint & spearmint), myrrh.

Varicose veins (S,C):

Cypress, lemon, lime, neroli, yarrow.

Verrucae (S,N):

Tagetes, tea tree.

Warts & corns (S,N):

Cinnamon leaf, lemon, lime, tagetes, tea tree.

Wounds (S,C,B):

Canadian balsam, Peru balsam, Tolu balsam, bergamot, cabreuva, chamomile (German & Roman), clove bud, cypress, elemi, eucalyptus (blue gum & lemon), frankincense, galbanum, geranium, immortelle, hyssop, juniper, lavandin, lavender (spike & true), linaloe, marigold, mastic, myrrh, niaouli, patchouli, rosewood, Levant styrax, tea tree, turpentine, vetiver, yarrow.

Wrinkles & mature skin (M,S,F,B):

Carrot seed, elemi, sweet fennel, frankincense, galbanum, geranium, jasmine, labdanum, true lavender, mandarin, mimosa, myrrh, neroli, palmarosa, patchouli, rose (cabbage & damask), rosewood, clary sage, sandalwood, spikenard, ylang ylang.... skin care

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