Also hyperglyconeogenesis. The state of excessive synthesis of glycogen (storage starch) or glucose by the liver, derived from non-sugar sources, such as amino acids, lactate and the glycerol remnants from triglyceride breakdown. In strictly subclinical terms it signifies a yinny, catabolic excess, wherein building materials are less desirable than FUEL, and it is singularly difficult to buff up in any way. There are disease states where this can occur...starvation would induce it as well, but I am not addressing this aspect, since I don’t consider this to be the realm of alternative approaches.
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