Insecticides Health Dictionary

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Insecticides: From 1 Different Sources

Substances which kill insects. Since the discovery of the insecticidal properties of DDT (see DICHLORODIPHENYL TRICHLOROETHANE) in 1940, a steady stream of new ones has been introduced. Their combined use has played an outstanding part in international public health campaigns, such as that of the World Health Organisation for the eradication of MALARIA.

Unfortunately, insects are liable to become resistant to insecticides, just as bacteria are liable to become resistant to antibiotics, and it is for this reason that so much research work is being devoted to the discovery of new ones. Researchers are also exploring new methods, such as releasing sexually sterile insects into the natural population.

The useful effects of insecticides must be set against increasing evidence that the indiscriminate use of some of these potent preparations is having an adverse e?ect – not only upon human beings, but also upon the ecosystems. Some, such as DDT – the use of which is now banned in the UK – are very stable compounds that enter the food chain and may ultimately be lethal to many animals, including birds and ?shes.

Health Source: Medical Dictionary
Author: Health Dictionary

Pesticides

Any substance or mixture of substances intended for preventing or controlling unwanted species of plants and animals. This includes any substances intended for use as plant-growth regulators, defoliants or desiccants. The main groups of pesticides are: herbicides to control weeds; insecticides to control insects; and fungicides to control or prevent fungal disease.... pesticides

Gulf War Syndrome

A group of symptoms caused by exposure of people in the armed forces to chemicals, including insecticides, used during the Gulf War in 1991.

Gulf War syndrome may also be due to side effects of vaccines and drugs given to the armed forces to prevent against possible attacks of chemical and biological weapons.... gulf war syndrome

Malaria

A parasitic disease caused by four species of PLASMODIUM: P. falciparum, P. vivax, P. ovale, and P. malariae. Clinically, malaria is characterised by recurrent episodes of high fever, sometimes associated with RIGOR; enlargement of the SPLEEN is common. P. falciparum infection can also be associated with several serious – often fatal – complications (see below): although other species cause chronic disease, death is unusual.

During a bite by the female mosquito, one or more sporozoites – a stage in the life-cycle of the parasite – are injected into the human circulation; these are taken up by the hepatocytes (liver cells). Following division, merozoites (minute particles resulting from the division) are liberated into the bloodstream where they invade red blood cells. These in turn divide, releasing further merozoites. As merozoites are periodically liberated into the bloodstream, they cause the characteristic fevers, rigors, etc.

Malaria occurs in many tropical and subtropical countries; P. falciparum is, however, con?ned very largely to Africa, Asia and South America. Malaria is present in increasingly large areas; in addition, the parasites are developing resistance to various preventative and treatment drugs. The disease constitutes a signi?cant problem for travellers, who must obtain sound advice on chemoprophylaxis before embarking on tropical trips – especially to a rural area where intense transmission can occur. Transmission has also been recorded at airports, and following blood transfusion.

The World Health Organisation (WHO) has listed malaria as one of Europe’s top ten infectious diseases. In 1992, 20,000 cases were reported: this had risen to more than 200,000 by the late 1990s. The resurgence of malaria has been worldwide, in part the result of the development of resistant strains of the disease, and in part because many countries have failed (or been unable) to implement environmental measures to eliminate mosquitoes. Nearly 40 years ago the WHO forecast that by 1980 only four million people would be affected worldwide; now, at the beginning of the 21st century, around 500 million people a year are contracting malaria with about 3,000 people a day dying from the infection – as many as 70 per cent of them children under the age of ?ve, according to WHO ?gures. The apparently steady advance of global warming means that countries with temperate climates may well warm up su?ciently to enable malaria to become established as an ENDEMIC disease. In any case, the great increase in international air travel has exposed many more people to the risk of malaria, and infected individuals may not exhibit symptoms until they are back home. Doctors seeing a recent traveller with unexplained pyrexia and illness should consider the possibility of malarial infection.

Diagnosis is by demonstration of trophozoites – a stage in the parasite’s life-cycle that takes place in red blood cells – in thick/thin blood-?lms of peripheral blood. Serological tests are of value in deciding whether an individual has had a past infection, but are of no value in acute disease.

P. vivax and P. ovale infections cause less severe disease than P. falciparum (see below), although overall there are many clinical similarities; acute complications are unusual, but chronic ANAEMIA is often present. Primaquine is necessary to eliminate the exoerythrocytic cycle in the hepatocyte (liver cell).

P. falciparum Complications of P. falciparum infection include cerebral involvement (see BRAIN – Cerebrum), due to adhesion of immature trophozoites on to the cerebral vascular endothelium; these lead to a high death rate when inadequately treated. Renal involvement (frequently resulting from HAEMOGLOBINURIA), PULMONARY OEDEMA, HYPOTENSION, HYPOGLYCAEMIA, and complications in pregnancy are also important. In complicated disease, HAEMODIALYSIS and exchange TRANSFUSION have been used. No adequate controlled trial using the latter regimen has been carried out, however, and possible bene?ts must be weighed against numerous potential side-effects – for instance, the introduction of a wide range of infections, overload of the circulatory system with infused ?uids, and other complications.

P. malariae usually produces a chronic infection, and chronic renal disease (nephrotic syndrome) is an occasional sequel, especially in tropical Africa.

Gross SPLENOMEGALY (hyper-reactive malarious splenomegaly, or tropical splenomegaly syndrome) can complicate all four human Plasmodium spp. infections. The syndrome responds to long-term malarial chemoprophylaxis. BURKITT’S LYMPHOMA is found in geographical areas where malaria infection is endemic; the EPSTEIN BARR VIRUS is aetiologically involved.

Prophylaxis Malaria specialists in the United Kingdom have produced guidance for residents travelling to endemic areas for short stays. Drug choice takes account of:

risk of exposure to malaria;

extent of drug resistance;

e?cacy of recommended drugs and their side-effects;

criteria relevant to the individual (e.g. age, pregnancy, kidney or liver impairment). Personal protection against being bitten by

mosquitoes is essential. Permethrinimpregnated nets are an e?ective barrier, while skin barrier protection and vaporised insecticides are helpful. Lotions, sprays or roll-on applicators all containing diethyltoluamide (DEET) are safe and work when put on the skin. Their e?ect, however, lasts only for a few hours. Long sleeves and trousers should be worn after dark.

Drug prophylaxis should be started at least a week before travelling into countries where malaria is endemic (two or three weeks in the case of me?oquine). Drug treatment should be continued for at least four weeks after leaving endemic areas. Even if all recommended antimalarial programmes are followed, it is possible that malaria may occur any time up to three months afterwards. Medical advice should be sought if any illness develops. Chloroquine can be used as a prophylactic drug where the risk of resistant falciparum malaria is low; otherwise, me?oquine or proguanil hydrochloride should be used. Travellers to malaria-infested areas should seek expert advice on appropriate prophylactic treatment well before departing.

Treatment Various chemoprophylactic regimes are widely used. Those commmonly prescribed include: chloroquine + paludrine, me?oquine, and Maloprim (trimethoprim + dapsone); Fansidar (trimethoprim + sulphamethoxazole) has been shown to have signi?cant side-effects, especially when used in conjunction with chloroquine, and is now rarely used. No chemotherapeutic regimen is totally e?ective, so other preventive measures are again being used. These include people avoiding mosquito bites, covering exposed areas of the body between dusk and dawn, and using mosquito repellents.

Chemotherapy was for many years dominated by the synthetic agent chloroquine. However, with the widespread emergence of chloroquine-resistance, quinine is again being widely used. It is given intravenously in severe infections; the oral route is used subsequently and in minor cases. Other agents currently in use include me?oquine, halofantrine, doxycycline, and the artemesinin alkaloids (‘qinghaosu’).

Researchers are working on vaccines against malaria.... malaria

Dichlorodiphenyl Trichloroethane

DDT is the generally used abbreviation for the compound which has been given the o?cial name of dicophane. It was ?rst synthesised in 1874, but it was not until 1940 that, as a result of research work in Switzerland, its remarkable toxic action on insects was discovered. This work was taken up and rapidly expanded in Great Britain and the USA, and one of its ?rst practical applications was in controlling the spread of TYPHUS FEVER. This disease is transmitted by the louse, one of the insects for which DDT is most toxic. Its toxic action against the mosquito has also been amply proved, and it thus rapidly became one of the most e?ective measures in controlling MALARIA. DDT is toxic to a large range of insects in addition to the louse and the mosquito; these include house?ies, bed-bugs, clothes-moths, ?eas, cockroaches, and ants. It is also active against many agricultural and horticultural pests, including weevils, ?our beetles, pine saw?y, and most varieties of scale insect.

DDT has thus had a wide use in medicine, public health, veterinary medicine, horticulture, and agriculture. Unfortunately, the indiscriminate use of DDT is potentially hazardous, and its use is now restricted or banned in several countries, including the United Kingdom.

The danger of DDT is that it enters the biological food chain with the result that animals at the end of the food chain such as birds or predators may build up lethal concentrations of the substance in their tissues.

In any case, an increasing number of species of insects were becoming resistant to DDT. Fortunately, newer insecticides have been introduced which are toxic to DDT-resistant insects, but there are doubts whether this supply of new insecticides can be maintained as insects develop resistance to them.... dichlorodiphenyl trichloroethane

Integrated Control

A combination of biological and insecticidal methods of control, e.g. the introduction of predacious fish to breeding places which are also sprayed with insecticides that have minimum effect on the fish.... integrated control

Juniperus Virginiana

Linn.

Family: Pinaceae; Cupressaceae.

Habitat: Native to North America; introduced into India.

English: Pencil Cedar, Red Cedar.

Action: The berries in decoction are diaphoretic and emmenagogue like those of common juniper; leaves are diuretic. Red cedar oil is used in the preparation of insecticides. Small excrescences, called cedar apples, are sometimes found on the branches. These are used as an anthelmintic. (Yellow Cedar is equated with Thuja occidentalles.)

Juniperus procera Hochst. (East African Cedar), J. bermudiana Linn. (Bermuda Cedar) andJ.ChinensisLinn. (Chinese Juniper) have also been introduced into India.... juniperus virginiana

Long Pepper

Piper longum

Piperaceae: San: Pippali;

Hin, Ben, Pun: Piplamul; Kan, Mal:Thippali ;

Tam: Thippili; Mar: Pimpli;

Tel: Pipppaloo; Ass: Piplu.

Introduction: Long pepper is a slender aromatic climber whose spike is widely used in ayurvedic and unani systems of medicine particularly for diseases of respiratory tract. Pipalarishta, Pippalyasava, Panchakola, Pippalayadilauha, and Lavana bhaskar churan are common ayurvedic preparations made out of the dry spikes of female types. Ittrifal fauladi, Angaruya-i-kabir and Majun khadar are well known unani preparations of long pepper. Its roots also have several medicinal uses. The root is useful in bronchitis, stomach ache, diseases of spleen and tumours. Fruit is useful in vata and kapha, asthma, bronchitis, abdominal complaints, fever, leucoderma, urinary discharges, tumours, piles, insomnia and tuberculosis. Root and fruit are used in gout and lumbago. The infusion of root is prescribed after parturition to induce the expulsion of placenta. The root and fruit decoction are used in acute and chronic bronchitis and cough. It contains the alkaloid piperine which has diverse pharmacological activities, including nerve depressant and antagonistic effect on electro- shock and chemo -shock seizures as well as muscular incoordination.

Distribution: The plant is a native of Indo-Malaya region. It was very early introduced to Europe and was highly regarded as a flavour ingredient by the Romans. The Greek name “Peperi”, the Latin “Piper” and the English “Pepper” were derived from the Sanskrit name “Pippali”. It grows wild in the tropical rain forests of India, Nepal, Indonesia, Malaysia, Sri lanka, Rhio, Timor and the Philippines. In India, it is seen in Assam, West Bengal, Uttar Pradesh, Madhya Pradesh, Maharashtra, Kerala, Karnataka.and Tamil Nadu. It is also cultivated in Bengal, Chirapunchi area of Assam, Akola-Amravati region of Maharashtra, Anamalai hills of Tamil Nadu, Orissa, Uduppi and Mangalore regions of Karnataka. Bulk of Indian long pepper comes from its wild growth in Assam, Shillong and West Bengal, supplemented by imports from Sri Lanka and Indonesia (Viswanathan,1995)

Botany: Piper longum Linn. is a member of Piperaceae family. The plant is a glabrous perennial under-shrub with erect or sub-scandent nodose stem and slender branches, the latter are often creeping or trailing and rooting below or rarely scandent reaching a few metres height. Leaves are simple, alternate, stipulate, and petiolate or nearly sessile; lower ones broadly ovate, cordate; upper ones oblong, oval, all entire, smooth, thin with reticulate venation; veins raised beneath. It flowers nearly throughout the year. Inflorescence is spike with unisexual small achlamydeous densely packed flowers and form very close clusters of small greyish green or darker grey berries. Female spikes with short thick stalk varying from 1.5 to2.5 cm in length and 0.5 to 0.7 cm in thickness.

A number of geographical races are available in different agroclimatic regions of India; the most popular being Assam, West Bengal and Nepal races. Piper officinarum DC; syn. Chavica officinarum Miquel, Piper pepuloides and Piper chaba Hunter are the other related species of importance.

Agrotechnology: Long pepper is a tropical plant adapted to high rainfall areas with high humidity. An elevation of 100-1000 m is ideal. It needs partial shade to the tune of 20-30% for best growth. The natural habitat of the plant is on the borders of streams. It is successfully cultivated in well drained forest soils rich in organic matter. Laterite soils with high organic matter content and moisture holding capacity are also suitable for cultivation.

Long pepper is propagated by suckers or rooted vine cuttings.15-20 cm long 3-5 nodded rooted vine cuttings establishes very well in polybags. The best time for raising nursery is March-April. Normal irrigation is given on alternate days. The rooted cuttings will be ready for transplanting in 2 months time. With the onset of monsoon in June the field is ploughed well and brought to good tilth. 15-20 cm raised beds of convenient length and breadth are taken. On these beds, pits are dug at 60 x 60 cm spacing and well decomposed organic manure at 100 g/pit is applied and mixed with the soil. Rooted vine cuttings from polybags are transplanted to these pits. Gap filling can be done after one month of planting.The crop needs heavy manuring at the rate of 20 t FYM/ha every year. Application of heavy dose organic matter and mulching increase water retention in the soil and control weeds. Small doses of chemical fertilisers can also be used. The crop needs irrigation once a week. Sprinkler irrigation is ideal. With irrigation the crop continues to produce spikes and off-season produce will be available. However, it is reported that unirrigated crop after the onset of monsoon grows vigorously and shows much hardiness than the irrigated crop.

Crop losses can be heavy due to pests and diseases. Mealy bugs and root grubs, attack the plant particularly during summer. Infested plants show yellowing and stunted growth. Application of systemic insecticides like nuvacron or dimecron will control the pests. Adults and nymphs of Helopeltis theivora severely feeds on the foliage which can be controlled by 0.25% neem kernel suspension. Rotting of leaves and vines during monsoon season is caused by Colletotrichum glorosporiodes and necrotic lesions and blights on the leaves during summer is caused by Colletotrichum and Cercospora spp. These diseases can be controlled by spraying of 1% Bordeaux mixture repeatedly. A virus like disease characterised by yellowing and crinkling of leaves, stunted growth and production of spikes of smaller size and inferior quality was also recently reported.

The vines start flowering six months after planting and flowers are produced almost throughout the year. The spikes mature in 2 months time. The optimum stage of harvest is when the spikes are blackish green. The pungency is highest at this stage. Spikes are hand picked when they become mature and then dried. The yield of dry spike is 400 kg /ha during first year, increases to 1000kg during third year and thereafter it decreases. Therefore, after 3 years the whole plant is harvested. The stem is cut close to the ground and roots are dug up. Average yield is 500 kg dry roots/ha (Viswanathan,1995).

Piper longum can also be cultivated as an intercrop in plantations of coconut, subabul and eucalyptus.

Post harvest technology: The harvested spikes are dried in sun for 4-5 days until they are perfectly dry. The green to dry spike ratio is 10:1.5 by weight. The dried spikes have to be stored in moisture proof containers. Stem and roots are cleaned, cut into pieces of 2.5-5 cm length, dried in shade and marketed as piplamool. There are three grades of piplamool, based on the thickness. The commercial drug consists 0.5-2.5 cm long ,0.5-2.5 mm thick, cylindrical pieces dirty light brown in colour and peculiar odour with a pungent bitter taste, producing numbness to the tongue.

Properties and activity: The spike of long pepper contains 4-5% piperine, piplartin, piperolactam, N-isobutyl deca trans-2-trans-4-dienamide and piporadione alkaloides, besides 0.7 % essential oil. Roots gave the alkaloids piperine, piperlongumine (piplartine) and piperlonguminine; sesamine, methyl 3, 4, 5-trimethoxy cinnamate. Stem gave triacoutane 22, 23 - dihydrostigmasterol. Fruit essential oil contains piperidine, caryophyllene and sesquiterpene alcohol (Atal et al, 1975).

The root is plungent, hot, stomachic, laxative, anthelmintic and carminative. The fruit is sweetish, pungent, hot, stomachic, aphrodisiac, alterative, laxative, antidysenteric, emmenagogue, abortifacient, diuretic and tonic. The essential oil is antimicrobial and anthelmintic.

N-isobutyl-deca-trans-2-trans-4-dienamide is antitubercular.

Piperine is hypotensive, antipyretic, analeptic, and nerve stimulant (Warrier et al, 1995).... long pepper

Neuritis

In?ammation affecting a nerve or nerves which may be localised to one part of the body – as, for instance, in SCIATICA – or which may be general, being then known as multiple neuritis, or POLYNEURITIS. Owing to the fact that the most peripheral parts of the nerves are usually affected in the latter condition (i.e. the ?ne subdivisions in the substance of the muscles), it is also known as peripheral neuritis.

Causes In cases of localised neuritis, the ?brous sheath of the nerve is usually at fault, the actual nerve-?bres being only secondarily affected. This condition may be due to in?ammation spreading into the nerve from surrounding tissues; to cold; or to long-continued irritation by pressure on the nerve. The symptoms produced vary according to the function of the nerve, in the case of sensory nerves being usually neuralgic pain (see NEURALGIA), and in the case of motor nerves some degree of paralysis in the muscles to which the nerves pass.

In polyneuritis, usually due to some general or constitutional cause, the nerve-?bres themselves in the small nerves degenerate and break down. The condition is protracted because, for recovery to occur, the growth of new nerve-?bres from the healthy part of the nerve has to take place. The cause of polyneuritis may be infection by a virus – for example, HERPES ZOSTER – or a bacterium, as in LEPROSY. Neuritis may also be the result of agents such as alcohol, lead or products from industrial or agricultural activities. ORGANOPHOSPHORUS insecticides are believed by some to be a factor in neuritis and other neurological conditions.... neuritis

Parathion

One of the ORGANOPHOSPHORUS insecticides. It is highly toxic to humans and must therefore be handled with the utmost care.... parathion

Permethrin

Along with phenothrin, this is a largely nontoxic pyrethroid insecticide, e?ective in SCABIES and lice infestations. Resistance may develop to these insecticides and also to MALATHION and CARBARYL, in which case topical treatment should be alternated among the di?erent varieties.... permethrin

Solanums

Solanum spp.

Solanaceae

Solanums comprise a very important group of medicinal plants having multifarious uses.

These plants belong to the family Solanaceae and genus Solanum. A number of species are reported to be medicinal which are briefly described below.

1. S. anguivi Lam. syn. S. indicum auct. non Linn.

Eng: Poison berry; San: Brhati, Simhi;

Hin: Barhauta, Birhatta;

Mal: Puthirichunda, Cheruchunda;

Tam: Karimulli, Puthirichundai;

Kan: Ramagulla;

Tel: Cittimulaga, Tellamulaka It is found throughout the tropics, in plains and at low elevations. It is much branched, very prickly undershrub, 0.3-1.5m in height. Leaves are simple, large, ovate, subentire, sinuate or lobed. Flowers are blue in extra-axillary cymes having stellately hairy and prickly peduncles. Fruits are globose berries, reddish or dark yellow with smooth or minutely pitted seeds. Its roots are useful in vitiated conditions of vata and kapha, odontalgia, dyspepsia, flatulence, colic, verminosis, diarrhoea, pruritus, leprosy, skin diseases, strangury, cough, asthma, bronchitis, amenorrhoea, dysmenorrhoea, fever, cardiac disorders and vomiting. Roots bitter, acrid, astringent, thermogenic, anodyne, digestive, carminative, anthelmintic, stomachic, constipating, resolvent, demulcent, depurative, diuretic, expectorant, aphrodisiac, emmenagogue, febrifuge and cardiotonic.

2. S. dulcamara Linn.

Eng: Bittersweet, Bitter night shade; San: Kakmachi; Pun: Rubabarik It is found in tropical situations in India and Sikkim. The plant is rich in alkaloidal glycosides like solamarine, tomatidenol, solasodine and soladulcine. The berry and twig are alterative, antisyphilitic, diaphoretic, resolvent, narcotic, diuretic, antirheumatic and used in liver disorders and psoriasis.

3. S. erianthum D. Don, syn. S. verbascifolium auct. non Linn.

San: Vidari;

Hin: Asheta;

Mal: Malachunda;

Tam: Malaichundai, Anaisundaikkai

Pun: Kalamena;

Tel: Rasagadi

The plant is distributed over the tropical and subtropical zones of India. The plant contains alkaloids and steroidal sapogenins. Leaves and fruits contain solasodine, solasodiene, solafloridine, diosgenin, vespertilin and pregnenolone. The plant is CNS depressant, antiinflammatory and useful in burns.

4. S. melongena Linn.

Eng: Brinjal, Egg plant; San: Varttaki;

Hin: Bengan, Badanjan;

Mal: Vazhuthina

Tam: Kattirikkai;

Kan: Badanekaya, Doddabadane;

Tel: Vankaya, Niruvanga

It is mainly cultivated as a vegetable throughout the tropics and subtropics. It is an erect or suffrutescent, herbaceous, armed or unarmed perennial shrub. Leaves are simple, large, entire and lobed. Flowers are blue, in clusters of 2-5. Fruits are large, white, yellow or dark purple berries of different shapes capped with thick persistent calyx. Seeds are many, yellow or cream and discoid. The roots, leaves and unripe fruits are useful in cholera, bronchitis, asthma, odontalgia and fever. The roots are laxative, analgesic and cardiotonic. Leaves are sialagogue, narcotic and antiherpetic. The unripe fruits are bitter, acrid, sweet, aphrodisiac, cardiotonic and haematinic.

5. S. melongena var. incanum (Linn.) Prain syn. S. incanum Linn., S. coagulens Forsk.

San: Brihati;

Hin: Baigan;

Mal: Cheruvazhuthina

It is a herbaceous prickly plant found in warm humid tropics. It is grown almost throughout the year in the plains and during summer on the hills. It grows 0.6-2m in height. Leaves are simple, alternate lobed. Flowers are blue or white, 5 lobed, calyx with spines. Fruits are ellipsoid berries. The plant is a constituent of the dasamoola which helps to overcome vitiated tridoshas and cures dyspepsia, fever, respiratory and cardiac disorders, skin ailments, vomiting, ulcers and poisonous affections. In Ayurveda the formulations like Brihatyadi Kashaya, dashamoolarishta, Indukantaghritam, Dasamoolaharithaki, etc are the important preparations with the roots. It is also used in the treatment of toothache and sore throat. The fruit is reported to stimulate the intrahepatic metabolism of cholesterol. Roots are antiasthmatic and stimulant. Leaves are used in cholera, bronchitis and asthma. Fruits are useful in liver complaints.

6. S. nigrum Linn. syn. S. rubrum Mill.

Eng: Black night shade; San: Kakamachi;

Hin: Makoy, Gurkkamai;

Mal: Karimthakkali;

Tam: Manathakkali, Milagutakkali;

Kan: Kakarndi;

Tel: Kamachi, Kachi

It is seen wild throughout India. It is an erect, divaricately branched, unarmed, suffrutescent annual herb. Leaves are ovate or oblong, sinuate-toothed or lobed and glabrous. Flowers are 3-8 in extra-axillary drooping subumbellate cymes. Fruits are purplish black or reddish berries. Seeds are many, discoid, yellow, minutely pitted. The whole plant is useful in vitiated conditions of tridosha, rheumatalgia, swellings, cough, asthma, bronchitis, wounds, ulcers, flatulence, dyspepsia, strangury, hepatomegaly, otalgia, hiccough, opthalmopathy, vomiting, cardiopathy, leprosy, skin diseases, fever, splenomegaly, haemarrhoids, nephropathy, dropsy and general debility. The plant is bitter, acrid, emollient, antiseptic, antiinflammatory, expectorant, anodyne, vulnerary, digestive, laxative, diuretic, cardiotonic, depurative, diaphoretic, febrifuge, rejuvenating, sedative, alterant and tonic.

7. S. spirale Roxb.

Hin: Munguskajur

It is seen wild in Assam and Khasi hills in India. Its root is diuretic and narcotic.

8. S. stramoniifolium Jacq., syn. S. ferox auct. non Linn.

San: Garbhada;

Hin: Rambaigan;

Mal: Anachunda;

Tam: Anaichundai;

Tel: Mulaka

It is observed in India in the states of Assam, Maharashtra, Karnataka and Tamil Nadu. Its berries contain glycoalkaloids such as solasonine and solasodine. Its roots and berries are bechic, antiasthmatic, antirheumatic, antiviral, anticancerous and spermicidal.

9. S. surattense Burm. F. syn. S. xanthocarpum schrad. & Wendl., S. jacquinii Willd.

Eng: Yellow-berried nightshade; San: Kantakari, Nidigdhika;

Hin: Remgani,Kateli;

Mal: Kantakarichunda;

Tam: Kantankattiri;

Kan: Nelagulli;

Tel: Callamulaga

It is found throughout India and Pakistan in dry situations as weed on roadsides and wastelands. It is prickly, diffuse, bright green, suffrutescent, perennial undershrub, with zigzag branches. Leaves are ovate-oblong, hairy on both sides and armed on the midrib and the nerves. Flowers are bluish purple, in extra-axillary cymes. Fruits are glabrous, globular drooping berry, yellow or white with green veins, surrounded by the calyx. Seeds are many, small, reniform, smooth and yellowish brown.

The whole plant is useful in vitiated conditions of vata and kapha, helminthiasis, dental caries, inflammations, flatulence, constipation, dyspepsia, anorexia, leprosy, skin diseases, hypertension, fever, cough, asthma, bronchitis, hiccough, lumbago, haemorrhoids and epilepsy. The plant is bitter, acrid, thermogenic, anthelmintic, antiinflammatory, anodyne, digestive, carminative, appetiser, stomachic, depurative, sudorific, febrifuge, expectorant, laxative, stimulant, diuretic, rejuvenating, emmenagogue and aphrodisac. Fruits contain solasonine, solamargine and solasodine.

10. S. torvum Sw.

Eng: West Indian Turkey Berry;

Hin,

Ben: Titbaigan;

Mal: Kattuchunda;

Kan: Kadu Sunde;

Tam: Sundaikai, Amarakai;

Tel: Kundavustic, Kotuvestu; Ass: Hathibhekuri

It is seen throughout tropical India, particularly in Orissa, Bihar and Manipur. The plant is CVS active and used in splenomegaly. Fruits and leaves contain solasonine, solasodine, jurubine, jurubidine, torvonin, torvogenin, chlorogenin, paniculogenin, sisalogenone, neosolaspigenin and solaspigenin.

11. S. trilobatum Linn.

Eng: Climbing Brinjal; San: Alarka;

Mal: Tutavalam;

Tam: Tuduvalai;

Kan: Mullumusta;

Tel: Telavuste

It is mostly seen in South and Western India. The plant contains alkamine and solamarine. The berry and flowers are bechic and used in bronchitis. The alkaloid solamarine is antibiotic and possesses antitumour activity.

12. S. viarum Dunal, syn. S. Khasianum C. B. Clarke

Hin: Kantakari

It is widely distributed in Khasi, Jaintia and Naga hills of Assam and Manipur upto 2000m and in Sikkim, West Bengal, Orissa and in the Niligiris. The plant and berries contain solasonine (which on hydrolysis yields solasodine), solamargine, khasianine, nantigenin, solasodine, diosgenin and saponin-solakhasianin. The plant is spasmolytic and CNS active. The berry is a source of solasodine used in the synthesis of corticosteroidal hormones.

Agrotechnology: The agrotechnology for the solanaceous group of plants are almost similar. They come up very well in tropical and subtropical climate upto 2000m altitude. They can be raised on a variety of soils good in organic matter. Propagation is by seeds. The seedlings are first raised in the nursery and transplanted to the main field 30-45 days after sowing when the plants attain 8-10cm height. During rainy season, planting is done on ridges while during summer in furrows, at a spacing ranging from 30-90cm depending upon the stature and spreading habit of the plant. The transplanted seedlings should be given temporary shade for 2-4 days during summer. FYM or compost at 20-25t/ha is applied at the time of land preparation. A moderate fertiliser dose of 75:40:40 N, P2O5, K2O/ha may be given. P is given as basal dose, N and K are applied in 2-3 split doses. One or two intercultural operations are needed to control weeds. The plants need earthing up after weeding and topdressing. Irrigation is needed at 3-4 days interval during summer and on alternate days during fruiting period. Plants need staking to avoid lodging due to heavy bearing. Shoot borers, mealy bugs, leaf webbers and miners are noted on the crop, which can be controlled by spraying mild insecticides. Root knot nematode, wilting and mosaic diseases are also noted on the crop. Field sanitation, crop rotation and burning of crop residues are recommended.... solanums

Bed Bug

a bloodsucking insect of the genus Cimex. C. hemipterus of the tropics and C. lectularius of temperate regions have reddish flattened bodies and vestigial wings. They live and lay their eggs in the crevices of walls and furniture and emerge at night to suck blood; although bed bugs are not known vectors of disease their bites leave a route for bacterial infection. Premises can be disinfested with appropriate insecticides.... bed bug

Organophosphorus

Organophosphorus insecticides act by inhibiting the action of cholinesterase (see ACETYLCHOLINE). For this reason they are also toxic to humans and must therefore be handled with great care. The most widely used are PARATHION and MALATHION. Organophosphorus has also been used to make nerve gases (see BIOLOGICAL WARFARE).

Treatment After contamination with insecticides, decontaminate (remove clothes, wash skin). Those treating should wear gloves, mask, apron and goggles. For symptoms give 2 mg of ATROPINE IV every 30 minutes until full atropinisation (dry mouth, pulse >70). Up to three days’ treatment may be needed. Severe poisoning may require pralidoxine mysalate: available from designated centres, this drug should be given intravenously within 24 hours of exposure.... organophosphorus

Osteoporosis

The softening of bone mass and the widening of the bone canals. This occurs with both age and diminished physical activity. Since women live longer, they are more likely to show such signs. (WARNING! Tirade Ahead!) There is little doubt that the condition is increasing among American women, and is starting to show itself at an earlier age. This is called “improved diagnostic methods” (harumph). The statistics that show the rise to be strongest in women that have used steroid hormone therapies in their earlier years seems to have escaped the notice of current Medical Conventional Wisdom. This states that ALL women need medical care against osteoporosis going into menopause, and the primary treatment is...steroid hormones (this year, at least). I know this may sound smarmy, coming from some long-in-the-tooth hippy male, but I would be far more impressed if SERIOUS attention was given to carefully defining the parameters of a woman’s risks. The road of medicine is strewn with four decades of well-intended universal hormone approaches to women’s health...embarrassedly forgotten. The idea of universal HRT for a whole generation of menopausal women seems like a frightening experiment in medical fascism and band-wagon hubris. There is no attention given as to WHY our future elders are suddenly stricken with a medical problem. Were birth-control pills, made up of synthetic digestion-proof steroid analogues, a major cause? Has our food become simply inadequate and over-pocessed? Have the decades of exposure by women to xeno-estrogens that are derived from degraded insecticides had more effect than the ones claimed by environmental watch-dog groups...the rise in breast and prostate cancer, the halving of the sperm count in Caucasian males and little-dicked alligators reported from Florida? Is the synthetic flavor in that pink bubble gum to blame? Perhaps its the fumes released from the early Barbies? FDS? There must be some reason, but the present medical answer is only HRT and (if politics allow) Jane Fonda tapes.... osteoporosis

Surveillance Of Disease

As distinct from surveillance of persons, surveillance of disease is the continuing scrutiny of all aspects of occurrences and spread of a disease that are pertinent to effective control. Included are the systematic collection and evaluation of: 1. morbidity and mortality reports; 2. special reports of field investigations, of epidemics and of individual cases; 3. isolation and identification of infectious agents by laboratories; 4. data concerning the availability and use of vaccines and toxoids, immunoglobulin, insecticides, and other substances used in control; 5. information regarding immunity levels in segments of the population; and 6. other relevant epidemiological data.... surveillance of disease

Bright’s Disease (acute)

Glomerulonephritis. Recognised by slight puffiness of the eyes and a dropsical accumulation of fluid in body cavities. Blood pressure rises. Appetite disappears. Digestion is deranged, urine may be blood-stained and a variety of symptoms present as dizziness, headache, nausea. Commonly caused by post streptococcal throat infection circulating in the blood, yet it is now known that the condition may arise from exposure to common garden insecticides and toxic substances of commercial importance that alter the body’s immune system and affect kidney function.

Acute toxic nephritis is possible in the convalescent stage of scarlet and other infectious fevers, even influenza. Causes are legion, including septic conditions in the ear, nose, throat, tonsils, teeth or elsewhere. Resistance to other infections will be low because of accumulation of toxins awaiting elimination. When protein escapes from the body through faulty kidneys general health suffers.

This condition should be treated by or in liaison with a qualified medical practitioner.

Treatment. Bedrest essential, with electric blanket or hot water bottle. Attention to bowels; a timely laxative also assists elimination of excessive fluid. Diuretics. Diaphoretics. Abundant drinks of bottled water or herb teas (3-5 pints daily). Alkaline drinks have a healing effect upon the kidneys. Juniper is never given for active inflammation.

Useful teas. Buchu, Cornsilk, Couchgrass, Clivers, Bearberry, Elderflowers, Marshmallow, Mullein, Marigold flowers, Wild Carrot, Yarrow.

Greece: traditional tea: equal parts, Agrimony, Bearberry, Couchgrass, Pellitory.

Powders. Equal parts: Dandelion, Cornsilk, Mullein. Dose: 750mg (three 00 capsules or half teaspoon) every 2 hours. In water or cup of Cornsilk tea.

Tinctures. Equal parts: Buchu, Elderflowers, Yarrow. Mix. Dose: 1-2 teaspoons in water or cup of Cornsilk tea, every two hours.

Topical. Hot poultices to small of the back; flannel or other suitable material saturated with an infusion of Elderflowers, Goldenrod, Horsetail or Yarrow. Herbal treatment offers a supportive role. ... bright’s disease (acute)

Disinfestation

n. the destruction of insect pests and other animal parasites. This generally involves the use of insecticides applied either topically, as in delousing, or as a spray for eliminating an infestation of fleas or bed bugs in the home.... disinfestation

Poisons

A poison is any substance which, if absorbed by, introduced into or applied to a living organism, may cause illness or death. The term ‘toxin’ is often used to refer to a poison of biological origin. Toxins are therefore a subgroup of poisons, but often little distinction is made between the terms. The study of the effects of poisons is toxicology and the effects of toxins, toxinology.

The concept of the dose-response is important for understanding the risk of exposure to a particular substance. This is embodied in a statement by Paracelsus (c.1493–1541): ‘All substances are poisons; there is none which is not a poison. The right dose di?erentiates a poison and a remedy.’

Poisoning may occur in a variety of ways: deliberate – SUICIDE, substance abuse or murder; accidental – including accidental overdose of medicines; occupational; and environmental

– including exposure during ?re.

Ingestion is the most common route of exposure, but poisoning may also occur through inhalation, absorption through the skin, by injection and through bites and stings of venomous animals. Poisoning may be described as acute, where a single exposure produces clinical effects with a relatively rapid onset; or chronic, where prolonged or repeated exposures may produce clinical effects which may be insidious in onset, cumulative and in some cases permanent.

Diagnosis of poisoning is usually by circumstantial evidence or elimination of other causes of the clinical condition of the patient. Some substances (e.g. opioids) produce a characteristic clinical picture in overdose that can help with diagnosis. In some patients laboratory analysis of body ?uids or the substance taken may be useful to determine or con?rm the o?ending agent. Routine assays are not necessary. For a very small number of poisons, such as paracetamol, aspirin, iron and lead, the management of the patient may depend on measuring the amount of poison in the bloodstream.

Accurate statistics on the incidence of poisoning in the UK are lacking. Mortality ?gures are more reliable than morbidity statistics; annually, well over 100,000 cases of poisoning are admitted to hospital. The annual number of deaths from poisoning is relatively small – about 300 – and in most cases patients die before reaching hospital. Currently, CARBON MONOXIDE (CO) is by far the most common cause of death due to poisoning. The most common agents involved in intentional or accidental poisoning are drugs, particularly ANALGESICS, ANTIDEPRESSANT DRUGS and SEDATIVES. Alcohol is also commonly taken by adults, usually in combination with drugs. Children frequently swallow household cleaners, white spirit, plant material – such as belladonna (deadly nightshade) and certain mushrooms; for example, death cap and ?y agaric – aftershave and perfume as well as drugs. If possible, the suspect container, drug or plant should be taken with the victim to the hospital or doctor. The use of child-resistant containers has reduced the number of admissions of children to hospital for treatment. Bixtrex® is an intensely bitter-tasting agent which is often added to products to discourage ingestion; however, not everybody is able to taste it, nor has any bene?cial e?ect been proven.

Treatment of poisoning usually begins with decontamination procedures. For ingested substances this may involve making the patient sick or washing the stomach out (GASTRIC LAVAGE): this is usually only worthwhile if performed soon after ingestion. It should be emphasised that salt (sodium chloride) water must never be given to induce vomiting, since this procedure is dangerous and has caused death. For substances spilt on the skin, the affected area should immediately be thoroughly washed and all contaminated clothing removed. Following eye exposure, the affected eye/s should be thoroughly irrigated with saline or water.

Treatment thereafter is generally symptomatic and supportive, with maintenance of the victim’s respiratory, neurological and cardiovascular systems and, where appropriate, monitoring of their ?uid and electrolyte balance and hepatic and renal function. There are speci?c antidotes for a few substances: the most important of these are PARACETAMOL, iron, cyanide (see CYANIDE POISONING), opioids (see OPIOID), DIGOXIN, insecticides and some heavy metals. Heavy-metal poisoning is treated with CHELATING AGENTS – chemical compounds that form complexes by binding metal ions: desferrioxamine and pencillinamine are two such agents. The number of people presenting with paracetamol overdose – a common drug used for attempted suicide – has fallen sharply since restrictions were placed on its over-thecounter sales.

When a patient presents with an illness thought to be caused by exposure to substances at work, further exposure should be limited or prevented and investigations undertaken to determine the source and extent of the problem. Acutely poisoned workers will usually go to hospital, but those suffering from chronic exposure may attend their GP with non-speci?c symptoms (see OCCUPATIONAL HEALTH, MEDICINE AND DISEASES).

In recent years, legislation has been enacted in the UK to improve safety in the workplace and to ensure that data on the hazardous constituents and effects of chemicals are more readily available. These o?cial controls include the Control of Substances Hazardous to Health (COSHH) and the Chemicals (Hazard Information and Packaging) Regulations (CHIP) and are UK legislation in response to European Union directives.

The National Poisons Information Service is a 24-hour emergency telephone service available to the medical profession and provides information on the likely effects of numerous agents and advice on the management of the poisoned patient. The telephone numbers are available in the medical literature. In the UK this is not a public-access service. People who believe they, or their relatives, have been poisoned should seek medical advice from their GPs or attend their local hospital.

Toxbase The National Poisons Information Service provides a primary clinical toxicology database on the Internet: www.spib.axl.co.uk. This website provides information about routine diagnosis, treatment and management of people exposed to drugs, household products and industrial and agricultural products.

(See also APPENDIX 1: BASIC FIRST AID.)... poisons

Typhus Fever

An infective disease of worldwide distribution, the manifestations of which vary in di?erent localities. The causative organisms of all forms of typhus fever belong to the genus RICKETTSIA. These are organisms which are intermediate between bacteria and viruses in their properties, and measure 0·5 micrometre or less in diameter.

Louse typhus, in which the infecting rickettsia is transmitted by the louse, is of worldwide distribution. More human deaths have been attributed to the louse via typhus, louse-borne RELAPSING FEVER and trench fever, than to any other insect with the exception of the MALARIA mosquito. Louse typhus includes epidemic typhus, Brill’s disease – which is a recrudescent form of epidemic typhus – and TRENCH FEVER.

Epidemic typhus fever, also known as exanthematic typhus, classical typhus, and louse-borne typhus, is an acute infection of abrupt onset which, in the absence of treatment, persists for 14 days. It is of worldwide distribution, but is largely con?ned today to parts of Africa. The causative organism is the Rickettsia prowazeki, so-called after Ricketts and Prowazek, two brilliant investigators of typhus, both of whom died of the disease. It is transmitted by the human louse, Pediculus humanus. The rickettsiae can survive in the dried faeces of lice for 60 days, and these infected faeces are probably the main source of human infection.

Symptoms The incubation period is usually 10–14 days. The onset is preceded by headache, pain in the back and limbs and rigors. On the third day the temperature rises, the headache worsens, and the patient is drowsy or delirious. Subsequently a characteristic rash appears on the abdomen and inner aspect of the arms, to spread over the chest, back and trunk. Death may occur from SEPTICAEMIA, heart or kidney failure, or PNEUMONIA about the 14th day. In those who recover, the temperature falls by CRISIS at about this time. The death rate is variable, ranging from nearly 100 per cent in epidemics among debilitated refugees to about 10 per cent.

Murine typhus fever, also known as ?ea typhus, is worldwide in its distribution and is found wherever individuals are crowded together in insanitary, rat-infested areas (hence the old names of jail-fever and ship typhus). The causative organism, Rickettsia mooseri, which is closely related to R. prowazeki, is transmitted to humans by the rat-?ea, Xenopsyalla cheopis. The rat is the main reservoir of infection; once humans are infected, the human louse may act as a transmitter of the rickettsia from person to person. This explains how the disease may become epidemic under insanitary, crowded conditions. As a rule, however, the disease is only acquired when humans come into close contact with infected rats.

Symptoms These are similar to those of louse-borne typhus, but the disease is usually milder, and the mortality rate is very low (about 1·5 per cent).

Tick typhus, in which the infecting rickettsia is transmitted by ticks, occurs in various parts of the world. The three best-known conditions in this group are ROCKY MOUNTAIN SPOTTED FEVER, ?èvre boutonneuse and tick-bite fever.

Mite typhus, in which the infecting rickettsia is transmitted by mites, includes scrub typhus, or tsutsugamushi disease, and rickettsialpox.

Rickettsialpox is a mild disease caused by Rickettsia akari, which is transmitted to humans from infected mice by the common mouse mite, Allodermanyssus sanguineus. It occurs in the United States, West and South Africa and the former Soviet Union.

Treatment The general principles of treatment are the same in all forms of typhus. PROPHYLAXIS consists of either avoidance or destruction of the vector. In the case of louse typhus and ?ea typhus, the outlook has been revolutionised by the introduction of e?cient insecticides such as DICHLORODIPHENYL TRICHLOROETHANE (DDT) and GAMMEXANE.

The value of the former was well shown by its use after World War II: this resulted in almost complete freedom from the epidemics of typhus which ravaged Eastern Europe after World War I, being responsible for 30 million cases with a mortality of 10 per cent. Now only 10,000–20,000 cases occur a year, with around a few hundred deaths. E?cient rat control is another measure which reduces the risk of typhus very considerably. In areas such as Malaysia, where the mites are infected from a wide variety of rodents scattered over large areas, the wearing of protective clothing is the most practical method of prophylaxis. CURATIVE TREATMENT was revolutionised by the introduction of CHLORAMPHENICOL and the TETRACYCLINES. These antibiotics altered the prognosis in typhus fever very considerably.... typhus fever

Anaemia, Aplastic

A rare but serious type of anaemia in which the red cells, white cells, and platelets in the blood are all reduced in number. Aplastic anaemia is caused by a failure of the bone marrow to produce stem cells, the initial form of all blood cells.

Treatment of cancer with radiotherapy or anticancer drugs can temporarily interfere with the cell-producing ability of bone marrow, as can certain viral infections and other drugs. Long-term exposure to insecticides or benzene fumes may cause more persistent aplastic anaemia, and a moderate to high dose of nuclear radiation is another recognized cause. An autoimmune disorder is responsible in about half of all cases. Aplastic anaemia sometimes develops for no known reason.

A low level of red blood cells may cause symptoms common to all types of anaemia, such as fatigue and breathlessness. White-cell deficiency increases susceptibility to infections; platelet deficiency may lead to a tendency to bruise easily, bleeding gums, and nosebleeds.

The disorder is usually suspected from blood-test results, particularly a blood count, and is confirmed by a bone marrow biopsy.

Blood and platelet transfusions can control symptoms.

Immunosuppression is used to treat anaemia due to an autoimmune process.

Severe persistent aplastic anaemia may be fatal unless a bone marrow transplant is carried out.... anaemia, aplastic

Filariasis

A group of tropical diseases, caused by various parasitic worms or their larvae, which are transmitted to humans by insect bites.

Some species of worm live in the lymphatic vessels. Swollen lymph nodes and recurring fever are early symptoms. Inflammation of lymph vessels results in localized oedema. Following repeated infections, the affected area, commonly a limb or the scrotum, becomes very enlarged and the skin becomes thick, coarse, and fissured, leading to a condition known as elephantiasis. The larvae of another type of worm invade the eye, causing blindness (see onchocerciasis). A third type, which may sometimes be seen and felt moving beneath the skin, causes loiasis, characterized by irritating and sometimes painful areas of oedema called calabar swellings.

The diagnosis of filariasis is confirmed by microscopic examination of the blood. The anthelmintic drugs diethylcarbamazine or ivermectin most often cure the infection but may cause side effects such as fever, sickness, muscle pains, and increased itching. Diethylcarbamazine can be given preventively, and the use of insecticides and protective clothing help to protect against insect bites. (See also roundworms; insects and disease.)... filariasis

Organophosphates

Highly poisonous agricultural insecticides that are harmful when absorbed through the skin, by inhalation, or by swallowing. Among the many possible symptoms are nausea, vomiting, abdominal cramps, diarrhoea, blurred vision, excessive sweating, headache, confusion, and twitching. Severe poisoning may cause breathing difficulty, palpitations, seizures, and unconsciousness. If left untreated, death may result.

Treatment may include washing out the stomach (see lavage, gastric) or removing soiled clothing and washing contaminated skin.

Injections of atropine may be given, and oxygen therapy and/or artificial ventilation may be needed.

With rapid treatment, people may survive doses that would otherwise have been fatal.

Long term effects of organophosphates in sheep dips are thought to be responsible for debilitating illness with neural, muscular, and mental symptoms.... organophosphates

Food Poisoning

an illness affecting the digestive system that results from eating food that is contaminated by bacteria or bacterial toxins, viruses, or (less commonly) by residues of insecticides (on fruit and vegetables) or poisonous chemicals such as lead or mercury. It can also be caused by eating poisonous fungi, berries, etc. Symptoms commence 1–24 hours after ingestion and include nausea, vomiting, diarrhoea, and abdominal pain. Food-borne infections are caused by bacteria of the genera *Salmonella, *Campylobacter, and *Listeria in foods of animal origin. The disease is transmitted by human carriers who handle the food, by shellfish growing in sewage-polluted waters, or by vegetables fertilized by manure. Toxin-producing bacteria causing food poisoning include those of the genus Staphylococcus, which rapidly multiply in warm foods; pathogenic *Escherichia coli; and the species Clostridium perfringens, which multiplies in reheated cooked meals. A rare form of food poisoning – *botulism – is caused by toxins produced by the bacterium Clostridium botulinum, which may contaminate badly preserved canned foods. See also gastroenteritis.... food poisoning

Citronella

Cymbopogon nardus

FAMILY: Poaceae (Gramineae)

SYNONYMS: Andropogon nardus, Sri Lanka citronella, Lenabatu citronella.

GENERAL DESCRIPTION: A tall, aromatic, perennial grass, which has derived from the wild-growing ‘managrass’ found in Sri Lanka.

DISTRIBUTION: Native to Sri Lanka, now extensively cultivated on the southernmost tip of the country.

OTHER SPECIES: An important essential oil is also produced on a large scale from the Java or Maha Pengiri citronella (C. winterianus). This variety is cultivated in the tropics worldwide, especially in Java, Vietnam, Africa, Argentina and Central America. There are many other related species of scented grasses.

HERBAL/FOLK TRADITION: The leaves of citronella are used for their aromatic and medicinal value in many cultures, for fever, intestinal parasites, digestive and menstrual problems, as a stimulant and an insect repellent. It is used in Chinese traditional medicine for rheumatic pain.

ACTIONS: Antiseptic, antispasmodic, bactericidal, deodorant, diaphoretic, diuretic, emmenagogue, febrifuge, fungicidal, insecticide, stomachic, tonic, vermifuge.

EXTRACTION: Essential oil by steam distillation of the fresh, part-dried or dried grass. (The Java citronella yields twice as much oil as the Sri Lanka type.)

CHARACTERISTICS: A yellowy-brown, mobile liquid with a fresh, powerful, lemony scent. The Java oil is colourless to pale yellow with a fresh, woody-sweet fragrance; it is considered of superior quality in perfumery work. It blends well with geranium, lemon, bergamot, orange, cedarwood and pine.

PRINCIPAL CONSTITUENTS: Mainly geraniol (up to 45 per cent in the Java oil), citronella (up to 50 per cent in the Java oil) with geranyl acetate, limonene and camphene, among others. The Sri Lanka variety contains more monoterpene hydrocarbons.

SAFETY DATA: Non-toxic, non-irritant; may cause dermatitis in some individuals. Avoid during pregnancy.

AROMATHERAPY/HOME: USE

Skin Care: Excessive perspiration, oily skin, insect repellant. ‘Mixed with cedarwood oil Virginia, it has been a popular remedy against mosquito attacks for many years prior to the appearance of DDT and other modern insecticides.’.

Immune System: Colds, ’flu, minor infections.

Nervous System: Fatigue, headaches, migraine, neuralgia.

OTHER USES: Extensively used in soaps, detergents, household goods and industrial perfumes. Employed in insect repellent formulations against moths, ants, fleas, etc, for use in the home and in the garden The Sri Lanka oil is used in most major food categories, including alcoholic and soft drinks. The Java oil is used as the starting material for the isolation of natural geraniol and citronellal.... citronella

Fennel

Foeniculum vulgare

FAMILY: Apiaceae (Umbelliferae)

SYNONYMS: F. officinale, F. capillaceum, Anethum foeniculum, fenkel.

GENERAL DESCRIPTION: Biennial or perennial herb up to 2 metres high, with feathery leaves and golden yellow flowers. There are two main varieties of fennel: bitter or common Fennel, slightly taller with less divided leaves occurring in a cultivated or wild form and sweet fennel (also known as Roman, garden or French fennel) which is always cultivated.

DISTRIBUTION: Bitter fennel is native to the Mediterranean region, found growing wild in France, Spain, Portugal and North Africa (they produce the ‘weed’oil). It is cultivated extensively worldwide, the main oil producers being Hungary, Bulgaria, Germany, France, Italy and India.

Sweet fennel is thought to have originated on the island of Malta, having been introduced by monks or crusaders thousands of years ago. It is now grown principally in France, Italy and Greece.

OTHER SPECIES: Bitter fennel (F. vulgare var. amara) and sweet fennel (F. vulgare var. dulce) are both closely related to the Florence fennel (F. azoricum), a smaller plant with a large cylindrical fleshy root which can be eaten as a vegetable. There are also many other cultivated varieties such as the German or Saxon fennel, the Russian, Indian and Japanese fennel, all of which produce slightly different oils.

HERBAL/FOLK TRADITION: A herb of ancient medical repute, believed to convey longevity, courage and strength. It was also used to ward off evil spirits, strengthen the eyesight and to neutralize poisons. In eastern and western herbalism it is considered good for obstructions of the liver, spleen and gall bladder and for digestive complaints such as colic, indigestion, nausea and flatulence (an ingredient of children’s ‘gripe water’).

It has traditionally been used for obesity, which may be due to a type of oestrogenic action, which also increases the milk of nursing mothers. Still current in the British Herbal Pharmacopoeia, used locally for conjunctivitis, blepharitis and pharyngitis.

ACTIONS: Aperitif, anti-inflammatory, antimicrobial, antiseptic, antispasmodic, carminative, depurative, diuretic, emmenagogue, expectorant, galactagogue, laxative, orexigenic, stimulant (circulatory), splenic, stomachic, tonic, vermifuge.

EXTRACTION: Essential oil by steam distillation. 1. Sweet fennel oil is obtained from crushed seeds, and 2. bitter fennel oil from crushed seeds or the whole herb (the wild ‘weed’).

CHARACTERISTICS: 1. A colourless to pale yellow liquid with a very sweet, anise-like, slightly earthy-peppery scent. It blends well with geranium, lavender, rose and sandalwood. 2. The seed oil is a pale yellow liquid with a sharp, warm camphoraceous odour; the ‘weed’ oil is pale orange-brown with a sharp, peppery-camphoraceous odour.

PRINCIPAL CONSTITUENTS: Anethole (50–60 per cent), limonene, phellandrene, pinene, anisic acid, anisic aldehyde, camphene, limonene, among others. In addition, bitter fennel oil contains 18–22 per cent fenchone, whereas the sweet fennel oil contains little or none.

SAFETY DATA: Non-irritant, relatively non-toxic, narcotic in large doses; bitter fennel may cause sensitization in some individuals. Sweet fennel oil is preferred in aromatherapy and perfumery work, since it does not contain the harsh ‘fenchone’ note, and because it is non-sensitizing. Bitter fennel oil should not be used on the skin at all, although it is considered superior medicinally. Neither oil should be used by epileptics or during pregnancy. Use in moderation.

AROMATHERAPY/HOME: USE Bitter fennel – none.

Sweet fennel:

Skin Care: Bruises, dull, oily, mature complexions, pyorrhoea.

Circulation Muscles And Joints: Cellulitis, obesity, oedema, rheumatism.

Respiratory System: Asthma, bronchitis.

Digestive System: Anorexia, colic, constipation, dyspepsia, flatulence, hiccough, nausea.

Genito-Urinary System: Amenorrhoea, insufficient milk (in nursing mothers), menopausal problems.

OTHER USES: In pharmaceutical products it is known as ‘codex’ fennel oil, used in cough drops, lozenges, etc; also used in carminative and laxative preparations. Extensively used as a flavour ingredient in all major food categories, in soft drinks and especially in alcoholic drinks such as brandy and liqueurs. Fennel oil (mainly sweet) is used in soaps, toiletries and perfumes. It also provides a good masking agent for industrial products, room sprays, insecticides, etc.... fennel

Lavender, Spike

Lavandula latifolia

FAMILY: Lamiaceae (Labiatae)

SYNONYMS: L. spica, aspic, broad-leaved lavender, lesser lavender, spike.

GENERAL DESCRIPTION: An aromatic evergreen sub-shrub up to 1 metre high with lance-shaped leaves, broader and rougher than true lavender. The flower is more compressed and of a dull grey-blue colour.

DISTRIBUTION: Native to the mountainous regions of France and Spain; also found in North Africa, Italy, Yugoslavia and the eastern Mediterranean countries. It is cultivated internationally; the oil is mainly produced in France and Spain.

OTHER SPECIES: There are many different chemotypes of lavender in general, and this also applies to spike lavender. The French spike oil is reputed to be a more delicate, aromatic scent than the Spanish variety. For other varieties, see entries on lavandin, true lavender and the Botanical Classification section.

HERBAL/FOLK TRADITION: Culpeper recommends spike lavender for a variety of ailments including ‘pains of the head and brain which proceed from cold, apoplexy, falling sickness, the dropsy, or sluggish malady, cramps, convulsions, palsies, and often faintings’. He also warns that ‘the oil of spike is of a fierce and piercing quality, and ought to be carefully used, a very few drops being sufficient for inward or outward maladies’.. The preparation ‘oleum spicae’ was made by mixing ¼ spike oil with ¾ turpentine, and used for paralysed limbs, old sprains and stiff joints (it was also said to encourage hair growth).

Spike lavender is current in the British Herbal Pharmacopoeia, indicated for flatulent dyspepsia, colic, depressive headaches, and the oil (topically) for rheumatic pain.

ACTIONS: See true lavender.

EXTRACTION: Essential oil by water or steam distillation from the flowering tops.

CHARACTERISTICS: A water-white or pale yellow liquid with a penetrating, fresh herbaceous, camphoraceous odour. It blends well with rosemary, sage, lavandin, eucalyptus, rosewood, lavender, petitgrain, pine, cedarwood, oakmoss, patchouli and spice oils, particularly clove.

PRINCIPAL CONSTITUENTS: Mainly cineol and camphor (40–60 per cent), with linalol and linalyl acetate, among others.

SAFETY DATA: Non-toxic, non-irritant (except in concentration), non-sensitizing.

AROMATHERAPY/HOME: USE See true lavender.

OTHER USES: It is used in some pharmaceutical preparations and especially in veterinary practice as a prophylactic, in incipient paralysis, for rheumatism and arthritis and to get rid of lice. It is extensively employed as a fragrance component especially in soaps and industrial perfumes such as deodorants, disinfectants and cleaning agents, as well as insecticides and room sprays, etc. It is also used in the food industry and in the production of fine varnishes and lacquers.... lavender, spike

Pine, Longleaf

Pinus palustris

FAMILY: Pinaceae

SYNONYMS: Longleaf yellow pine, southern yellow pine, pitch pine, pine (oil).

GENERAL DESCRIPTION: A tall evergreen tree with long needles and a straight trunk, grown extensively for its timber. It exudes a natural oleoresin from the trunk, which provides the largest source for the production of turpentine in America – see also entry on turpentine.

DISTRIBUTION: Native to south eastern USA, where the oil is largely produced.

OTHER SPECIES: There are numerous other species of pine all over the world which are used to produce pine oil, as well as pine needle and turpentine oil – see Botanical Classification section.

HERBAL/FOLK TRADITION: Pine sawdust has been used for centuries as a highly esteemed household remedy for a variety of ailments. ‘It is a grand, gentle, although powerful external antiseptic remedy, applied as a poultice in rheumatism when localised, hard cancerous tumours, tuberculosis in the knee or ankle joints, disease of the bone, in short, all sluggish morbid deposits ... I have used it behind the head for failing sight, down the spine for general debility, on the loins for lumbago, etc. all with the best results.’.

ACTIONS: Analgesic (mild), antirheumatic, antiseptic, bactericidal, expectorant, insecticidal, stimulant.

EXTRACTION: The crude oil is obtained by steam distillation from the sawdust and wood chips from the heartwood and roots of the tree (wastage from the timber mills), and then submitted to fractional distillation under atmospheric pressure to produce pine essential oil.

CHARACTERISTICS: A water-white or pale yellow liquid with a sweet-balsamic, pinewood scent. It blends well with rosemary, pine needle, cedarwood, citronella, rosewood, ho leaf and oakmoss.

PRINCIPAL CONSTITUENTS: Terpineol, estragole, fenchone, fenchyl alcohol and borneol, among others.

SAFETY DATA: Non-toxic; non-irritant (except in concentration); possible sensitization in some individuals.

AROMATHERAPY/HOME: USE

Circulation muscles and joints: Arthritis, debility, lumbago, muscular aches and pains, poor circulation, rheumatism, stiffness, etc.

Respiratory system: Asthma, bronchitis, catarrh, sinusitis.

OTHER USES: Used extensively in medicine, particularly in veterinary antiseptic sprays, disinfectants, detergents and insecticides (as a solvent carrier). Employed as a fragrance component in soaps, toiletries, bath products and perfumes. Also used in paint manufacture although it is increasingly being replaced by synthetic ‘pine oil’.... pine, longleaf

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