Nutritional Profile Energy value (calories per serving): Low Protein: Low Fat: Low Saturated fat: Low Cholesterol: None Carbohydrates: High Fiber: Low Sodium: Moderate Major vitamin contribution: Vitamin C Major mineral contribution: Iron, potassium
About the Nutrients in This Food Cranberries are nearly 90 percent water. The rest is sugars and dietary fiber, including insoluble cellulose in the skin and soluble gums and pectins in the flesh. Pectin dissolves as the fruit ripens; the older and riper the cran- berries, the less pectin they contain. Cranberries also have a bit of protein and a trace of fat, plus moderate amounts of vitamin C. One-half cup cranberries has 1.6 g dietary fiber and 6.5 mg vitamin C (9 percent of the R DA for a woman, 7 percent of the R DA for a man). One-half cup cranberry sauce has 1.5 g dietary fiber and 3 mg vitamin C (4 percent of the R DA for a woman, 3 percent of the R DA for a man).
The Most Nutritious Way to Serve This Food Relish made of fresh, uncooked berries (to preserve the vitamin C, which is destroyed by heat) plus oranges.
Diets That May Restrict or Exclude This Food Low-fiber diet
Buying This Food Look for: Firm, round, plump, bright red berries that feel cool and dry to the touch. Avoid: Shriveled, damp, or moldy cranberries. Moldy cranberries may be contaminated with fusarium molds, which produce toxins that can irritate skin and damage tissues by inhibiting the synthesis of DNA and protein.
Storing This Food Store packaged cranberries, unwashed, in the refrigerator, or freeze unwashed berries in sealed plastic bags for up to one year.
Preparing This Food Wash the berries under running water, drain them, and pick them over carefully to remove shriveled, damaged, or moldy berries. R inse frozen berries. It is not necessary to thaw before cooking.
What Happens When You Cook This Food First, the heat will make the water inside the cranberry swell, so that if you cook it long enough the berry will burst. Next, the anthocyanin pigments that make cranberries red will dissolve and make the cooking water red. Anthocyanins stay bright red in acid solutions and turn bluish if the liquid is basic (alkaline). Cooking cranberries in lemon juice and sugar preserves the color as well as brightens the taste. Finally, the heat of cooking will destroy some of the vitamin C in cranberries. Cranberry sauce has about one-third the vitamin C of an equal amount of fresh cranberries.
Medical Uses and/or Benefits Urinary antiseptic. Cranberr y juice is a long-honored folk remedy for urinar y infections. In 1985, researchers at Youngstown (Ohio) State University found a “special factor” in cran- berries that appeared to keep disease-causing bacteria from adhering to the surface of cells in the bladder and urinar y tract. In 1999, scientists at study at Rutgers University (in New Jersey) identified specific tannins in cranberries as the effective agents. In 2004, research- ers at Beth Israel Medical Center (New York) published a review of 19 recent studies of cranberries. The report, in the journal American Family Physician, suggested that a regimen of eight ounces of unsweetened cranberr y juice or one 300 – 400 mg cranberr y extract tablet twice a day for up to 12 months safely reduced the risk of urinar y tract infections. In 2008, a similar review by scientists at the University of Stirling (Scotland) of 10 studies showed similar results.
Adverse Effects Associated with This Food Increased risk of kidney stones. Long-term use of cranberry products may increase the risk of stone formation among patients known to form oxalate stones (stones composed of calcium and/or other minerals).
Food/Drug Interactions Anticoagulants Anticoagulants (blood thinners) are drugs used to prevent blood clots. They are most commonly prescribed for patients with atrial fibrillation, an irregular heartbeat that allows blood to pool in the heart and possibly clot before being pumped out into the body. In 2006 researchers at the College of Pharmacy and the Antithrombosis Center at the Univer- sity of Illinois (Chicago) reported that consuming cranberry juice while using the anticoagu- lant warafin (Coumadin) might cause fluctuations in blood levels of the anticoagulant, thus reducing the drug’s ability to prevent blood clots.... cranberries
Insulin-dependent and non-insulindependent diabetes have a varied pathological pattern and are caused by the interaction of several genetic and environmental factors.
Insulin-dependent diabetes mellitus (IDDM) (juvenile-onset diabetes, type 1 diabetes) describes subjects with a severe de?ciency or absence of insulin production. Insulin therapy is essential to prevent KETOSIS – a disturbance of the body’s acid/base balance and an accumulation of ketones in the tissues. The onset is most commonly during childhood, but can occur at any age. Symptoms are acute and weight loss is common.
Non-insulin-dependent diabetes mellitus (NIDDM) (maturity-onset diabetes, type 2 diabetes) may be further sub-divided into obese and non-obese groups. This type usually occurs after the age of 40 years with an insidious onset. Subjects are often overweight and weight loss is uncommon. Ketosis rarely develops. Insulin production is reduced but not absent.
A new hormone has been identi?ed linking obesity to type 2 diabetes. Called resistin – because of its resistance to insulin – it was ?rst found in mice but has since been identi?ed in humans. Researchers in the United States believe that the hormone may, in part, explain how obesity predisposes people to diabetes. Their hypothesis is that a protein in the body’s fat cells triggers insulin resistance around the body. Other research suggests that type 2 diabetes may now be occurring in obese children; this could indicate that children should be eating a more-balanced diet and taking more exercise.
Diabetes associated with other conditions (a) Due to pancreatic disease – for example, chronic pancreatitis (see PANCREAS, DISORDERS OF); (b) secondary to drugs – for example, GLUCOCORTICOIDS (see PANCREAS, DISORDERS OF); (c) excess hormone production
– for example, growth hormone (ACROMEGALY); (d) insulin receptor abnormalities; (e) genetic syndromes (see GENETIC DISORDERS).
Gestational diabetes Diabetes occurring in pregnancy and resolving afterwards.
Aetiology Insulin-dependent diabetes occurs as a result of autoimmune destruction of beta cells within the PANCREAS. Genetic in?uences are important and individuals with certain HLA tissue types (HLA DR3 and HLA DR4) are more at risk; however, the risks associated with the HLA genes are small. If one parent has IDDM, the risk of a child developing IDDM by the age of 25 years is 1·5–2·5 per cent, and the risk of a sibling of an IDDM subject developing diabetes is about 3 per cent.
Non-insulin-dependent diabetes has no HLA association, but the genetic in?uences are much stronger. The risks of developing diabetes vary with di?erent races. Obesity, decreased exercise and ageing increase the risks of disease development. The risk of a sibling of a NIDDM subject developing NIDDM up to the age of 80 years is 30–40 per cent.
Diet Many NIDDM diabetics may be treated with diet alone. For those subjects who are overweight, weight loss is important, although often unsuccessful. A diet high in complex carbohydrate, high in ?bre, low in fat and aiming towards ideal body weight is prescribed. Subjects taking insulin need to eat at regular intervals in relation to their insulin regime and missing meals may result in hypoglycaemia, a lowering of the amount of glucose in the blood, which if untreated can be fatal (see below).
Oral hypoglycaemics are used in the treatment of non-insulin-dependent diabetes in addition to diet, when diet alone fails to control blood-sugar levels. (a) SULPHONYLUREAS act mainly by increasing the production of insulin;
(b) BIGUANIDES, of which only metformin is available, may be used alone or in addition to sulphonylureas. Metformin’s main actions are to lower the production of glucose by the liver and improve its uptake in the peripheral tissues.
Complications The risks of complications increase with duration of disease.
Diabetic hypoglycaemia occurs when amounts of glucose in the blood become low. This may occur in subjects taking sulphonylureas or insulin. Symptoms usually develop when the glucose concentration falls below 2·5 mmol/l. They may, however, occur at higher concentrations in subjects with persistent hyperglycaemia – an excess of glucose – and at lower levels in subjects with persistent hypo-glycaemia. Symptoms include confusion, hunger and sweating, with coma developing if blood-sugar concentrations remain low. Re?ned sugar followed by complex carbohydrate will return the glucose concentration to normal. If the subject is unable to swallow, glucagon may be given intramuscularly or glucose intravenously, followed by oral carbohydrate, once the subject is able to swallow.
Although it has been shown that careful control of the patient’s metabolism prevents late complications in the small blood vessels, the risk of hypoglycaemia is increased and patients need to be well motivated to keep to their dietary and treatment regime. This regime is also very expensive. All risk factors for the patient’s cardiovascular system – not simply controlling hyperglycaemia – may need to be reduced if late complications to the cardiovascular system are to be avoided.
Diabetes is one of the world’s most serious health problems. Recent projections suggest that the disorder will affect nearly 240 million individuals worldwide by 2010 – double its prevalence in 1994. The incidence of insulin-dependent diabetes is rising in young children; they will be liable to develop late complications.
Although there are complications associated with diabetes, many subjects live normal lives and survive to an old age. People with diabetes or their relatives can obtain advice from Diabetes UK (www.diabetes.org.uk).
Increased risks are present of (a) heart disease, (b) peripheral vascular disease, and (c) cerebrovascular disease.
Diabetic eye disease (a) retinopathy, (b) cataract. Regular examination of the fundus enables any abnormalities developing to be detected and treatment given when appropriate to preserve eyesight.
Nephropathy Subjects with diabetes may develop kidney damage which can result in renal failure.
Neuropathy (a) Symmetrical sensory polyneuropathy; damage to the sensory nerves that commonly presents with tingling, numbness of pain in the feet or hands. (b) Asymmetrical motor diabetic neuropathy, presenting as progressive weakness and wasting of the proximal muscles of legs. (c) Mononeuropathy; individual motor or sensory nerves may be affected. (d) Autonomic neuropathy, which affects the autonomic nervous system, has many presentations including IMPOTENCE, diarrhoea or constipation and postural HYPOTENSION.
Skin lesions There are several skin disorders associated with diabetes, including: (a) necrobiosis lipoidica diabeticorum, characterised by one or more yellow atrophic lesions on the legs;
(b) ulcers, which most commonly occur on the feet due to peripheral vascular disease, neuropathy and infection. Foot care is very important.
Diabetic ketoacidosis occurs when there is insu?cient insulin present to prevent KETONE production. This may occur before the diagnosis of IDDM or when insu?cient insulin is being given. The presence of large amounts of ketones in the urine indicates excess ketone production and treatment should be sought immediately. Coma and death may result if the condition is left untreated.
Symptoms Thirst, POLYURIA, GLYCOSURIA, weight loss despite eating, and recurrent infections (e.g. BALANITIS and infections of the VULVA) are the main symptoms.
However, subjects with non-insulindependent diabetes may have the disease for several years without symptoms, and diagnosis is often made incidentally or when presenting with a complication of the disease.
Treatment of diabetes aims to prevent symptoms, restore carbohydrate metabolism to as near normal as possible, and to minimise complications. Concentration of glucose, fructosamine and glycated haemoglobin in the blood are used to give an indication of blood-glucose control.
Insulin-dependent diabetes requires insulin for treatment. Non-insulin-dependent diabetes may be treated with diet, oral HYPOGLYCAEMIC AGENTS or insulin.
Insulin All insulin is injected – mainly by syringe but sometimes by insulin pump – because it is inactivated by gastrointestinal enzymes. There are three main types of insulin preparation: (a) short action (approximately six hours), with rapid onset; (b) intermediate action (approximately 12 hours); (c) long action, with slow onset and lasting for up to 36 hours. Human, porcine and bovine preparations are available. Much of the insulin now used is prepared by genetic engineering techniques from micro-organisms. There are many regimens of insulin treatment involving di?erent combinations of insulin; regimens vary depending on the requirements of the patients, most of whom administer the insulin themselves. Carbohydrate intake, energy expenditure and the presence of infection are important determinants of insulin requirements on a day-to-day basis.
A new treatment for diabetes, pioneered in Canada and entering its preliminary clinical trials in the UK, is the transplantation of islet cells of Langerhans from a healthy person into a patient with the disorder. If the transplantation is successful, the transplanted cells start producing insulin, thus reducing or eliminating the requirement for regular insulin injections. If successful the trials would be a signi?cant advance in the treatment of diabetes.
Scientists in Israel have developed a drug, Dia Pep 277, which stops the body’s immune system from destroying pancratic ? cells as happens in insulin-dependent diabetes. The drug, given by injection, o?ers the possibility of preventing type 1 diabetes in healthy people at genetic risk of developing the disorder, and of checking its progression in affected individuals whose ? cells are already perishing. Trials of the drug are in progress.... diabetes mellitus
Jessabell, Jetzabel, Jezabel, Jezabella, Jezebelle, Jezibel, Jezibelle, Jezybell, Jezabella... jezebel
Meerah, Meira, Meirah, Meir... meera
Miah, Mea, Meah, Meya... mia
Nutritional Profile Energy value (calories per serving): Low Protein: Low Fat: Low Saturated fat: Low Cholesterol: None Carbohydrates: High Fiber: Moderate Sodium: Low Major vitamin contribution: Vitamin C Major mineral contribution: Calcium
About the Nutrients in This Food Blueberries have some protein and a little fat. They have no starch but do contain sugars and dietary fiber—primarily pectin, which dissolves as the fruit matures—and lignin in the seeds. (The difference between blueber- ries and huckleberries is the size of their seeds; blueberries have smaller ones than huckleberries.) One-half cup fresh blueberries has 1.5 g dietary fiber and 9.5 mg. vitamin C (13 percent of the R DA for a woman, 11 percent of the R DA for a man).
The Most Nutritious Way to Serve This Food Fresh, raw, or lightly cooked.
Buying This Food Look for: Plump, firm dark-blue berries. The whitish color on the ber- ries is a natural protective coating. Avoid: Baskets of berries with juice stains or liquid leaking out of the berries. The stains and leaks are signs that there are crushed (and possibly moldy) berries inside.
Storing This Food Cover berries and refrigerate them. Then use them in a day or two. Do not wash berries before storing. The moisture increases the chance that they will mold in the refrigerator. Also, handling the berries can damage them, tearing cells and releas- ing enzymes that will destroy vitamins. Do not store blueberries in metal containers. The anthocyanin pigments in the berries can combine with metal ions to form dark, unattractive pigment/metal compounds that stain the containers and the berries.
Preparing This Food R inse the berries under cool running water, then drain them and pick them over carefully to remove all stems, leaves, and hard (immature) or soft (over-ripe) berries.
What Happens When You Cook This Food Cooking destroys some of the vitamin C in fresh blueberries and lets water-soluble B vitamins leach out. Cooked berries are likely to be mushy because heat dissolves the pectin inside. Blueberries may also change color when cooked. The berries are colored with blue anthocyanin pigments. Ordinarily, anthocyanin-pigmented fruits and vegetables turn red- dish in acids (lemon juice, vinegar) and deeper blue in bases (baking soda). But blueberries also contain yellow pigments (anthoxanthins). In a basic (alkaline) environments, as in a batter with too much baking soda, the yellow and blue pigments will combine, turning the blueberries greenish blue. Adding lemon juice to a blueberry pie stabilizes these pigments; it is a practical way to keep the berries a deep, dark reddish blue.
How Other Kinds of Processing Affect This Food Canning and freezing. The intense heat used in canning the fruit or in blanching it before freezing reduces the vitamin C content of blueberries by half.
Medical Uses and/or Benefits Anticancer activity. According to the U.S. Department of Agriculture, wild blueberries rank first among all fruits in antioxidant content; cultivated blueberries (the ones sold in most food markets) rank second. Antioxidants are natural chemicals that inactivate free radicals, molecule fragments that can link together to form cancer-causing compounds. Several ani- mal studies attest to the ability of blueberries to inhibit the growth of specific cancers. For example, in 2005, scientists at the University of Georgia reported in the journal Food Research International that blueberry extracts inhibited the growth of liver cancer cells in laboratory settings. The following year, researchers at Rutgers University (in New Jersey) delivered data to the national meeting of the American Chemical Society from a study in which laboratory rats fed a diet supplemented with pterostilbene, another compound extracted from blueber- ries, had 57 percent fewer precancerous lesions in the colon than rats whose diet did not contain the supplement. The findings, however, have not been confirmed in humans. Enhanced memory function. In 2008, British researchers at the schools of Food Biosciences and Psychology at the University of Reading and the Institute of Biomedical and Clinical Sciences at the Peninsula Medical School (England) reported that adding blueberries to one’s normal diet appears to improve both long-term and short-term memory, perhaps because anthocyanins and flavonoids (water-soluble pigments in the berries) activate signals in the hippocampus, a part of the brain that controls learning and memory. If confirmed, the data would support the role played by diet in maintaining memory and brain function. Urinary antiseptic. A 1991 study at the Weizmann Institute of Science (Israel) suggests that blueberries, like cr anber r ies, contain a compound that inhibits the ability of Escherichia coli, a bacteria commonly linked to urinary infections, to stick to the wall of the bladder. If it cannot cling to cell walls, the bacteria will not cause an infection. This discovery lends some support to folk medicine, but how the berries work, how well they work, or in what “dos- ages” remains to be proven.
Adverse Effects Associated with This Food Allergic reaction. Hives and angiodemea (swelling of the face, lips, and eyes) are common allergic responses to berries, virtually all of which have been reported to trigger these reac- tions. According to the Merck Manual, berries are one of the 12 foods most likely to trigger classic food allergy symptoms. The others are chocolate, corn, eggs, fish, legumes (peas, lima beans, peanuts, soybeans), milk, nuts, peaches, pork, shellfish, and wheat (see wheat cer ea ls).... blueberries
Habitat: Throughout the drier and subtropical parts of India.
English: Winter Cherry. (Physalis alkekengi is also known as Winter Cherry.)Ayurvedic: Ashwagandhaa, Haya- gandhaa, Ashwakanda, Gandharva- gandhaa, Turaga, Turagagandhaa, Turangagandhaa, Vaajigandhaa, Gokarnaa, Vrishaa, Varaahakarni, Varadaa, Balyaa, Vaajikari. (A substitute for Kaakoli and Kshira- kaakoli.) Cultivated var.: Asgandh Naagori. (Indian botanists consider the cultivated plants distinct from the wild ones.)Unani: Asgandh.Siddha: Amukkuramkizhangu.Action: Root—used as an antiinflammatory drug for swellings, tumours, scrofula and rheumatism; and as a sedative and hypnotic in anxiety neurosis. Leaf— anti-inflammatory, hepatopro- tective, antibacterial. Fruits and seeds—diuretic. Withanine— sedative, hypnotic. Withaferin A—major component of biologically active steroids; as effective as hydrocortisone dose for dose. Antibacterial, antitumour, an- tiarthritic, significantly protective against hepatotoxicity in rats.
The root contains several alkaloids, including withanine, withananine, withananinine, pseudo-withanine, somnine, somniferine, somniferinine. The leaves of Indian chemotype contain 12 withanolides, including withaferin A. Steroidal lactones ofwithano- lide series have been isolated.Withanine is sedative and hypnotic. Withaferin A is antitumour, an- tiarthritic and antibacterial. Anti-inflammatory activity has been attributed to biologically active steroids, of which withaferin A is a major component. The activity is comparable to that of hydrocortisone sodium succinate.Withaferin A also showed significantly protective effect against CCl4- induced hepatotoxicity in rats. It was as effective as hydrocortisone dose for dose.The root extract contains an ingredient which has GABA mimetic activityThe free amino acids present in the root include aspartic acid, glycine, tyrosine, alanine, proline, tryptophan, glutamic acid and cystine.The Ayurvedic Pharmacopoeia ofIn- dia recommends Ashwagandha in im- potency. This claim could not be sustained in a recent experiment and raises a doubt about the equation of classical Ashwagandha with Withania somnifera. A methanolic extract of With- ania somnifera root induced a marked impairment in libido, sexual performance, sexual vigour and penile dysfunction in male rats. (Llayperuma et al, Asian J Androl, 2002, 295-298.)The total alkaloids of the root exhibited prolonged hypotensive, brady- cardiac and depressant action of the higher cerebral centres in several experimental animals.A withanolide-free aqueous fraction isolated from the roots of Withania somnifera exhibited antistress activity in a dose-dependent manner in mice. (Phytother Res 2003, 531-6.)(See also Simon Mills; American Herbal Pharmacopoeia, 2000; Natural Medicines Comprehensive Database, 2007.)Dosage: Root—3-6 g powder. (API, Vol. I.)... withania ashwagandhaFAMILY: Rosaceae
SYNONYMS: P. amygdalus var. amara, Amygdalus communis var. amara, A. dulcis, P. communis.
GENERAL DESCRIPTION: The almond tree grows to a height of about 7 metres and is popular as a garden tree due to its pinky-white blossom. It is botanically classified as a drupe.
DISTRIBUTION: Native to Western Asia and North Africa, it is now extensively cultivated throughout the Mediterranean region, Israel and California.
OTHER SPECIES: There are two main types of almond tree – bitter and sweet. The sweet almond does not produce any essential oil.
HERBAL/FOLK TRADITION: A ‘fixed’ oil commonly known as ‘sweet almond oil’ is made by pressing the kernels from both the sweet and bitter almond trees. Unlike the essential oil, this fixed oil does not contain any benzaldehyde or prussic acid, and has many medical and cosmetic uses. It is used as a laxative, for bronchitis, coughs, heartburn and for disorders of the kidneys, bladder and biliary ducts. It helps relieve muscular aches and pains, softens the skin and premotes a clear complexion.
ACTIONS: Anaesthetic, antispasmodic, narcotic, vermifuge (FFPA).
EXTRACTION: Essential oil by steam distillation from the kernels. The nuts are first pressed and macerated in warm water for 12 to 24 hours before the oil is extracted. It is during this process that the prussic acid is formed; it is not present in the raw seed. Most commercial bitter almond oil is rectified to remove all prussic acid, i.e. free from prussic acid (FFPA).
CHARACTERISTICS: Light colourless liquid with a characteristic ‘marzipan’ scent (FFPA).
PRINCIPAL CONSTITUENTS: Benzaldehyde (95 per cent), prussic acid (3 per cent).
SAFETY DATA: Prussic acid, also known as hydrocyanic acid or cyanide, is a well-known poison. Benzaldehyde is also moderately toxic.
AROMATHERAPY/HOME: USE None. ‘Should not be used in therapy either internally or externally.’2
OTHER USES: Bitter almond oil is no longer used for internal medication. Rectified bitter almond oil is used for flavouring foods, mainly confectionery; the most common uses are ‘almond essence’ and marzipan. The oil (FFPA) is increasingly being replaced by synthetic benzaldehyde in food flavourings.... almond, bitter
FAMILY: Asteraceae (Compositae)
SYNONYMS: O. mixta, Anthemis mixta, Moroccan chamomile.
GENERAL DESCRIPTION: A handsome plant, 90 to 125 cms high with very hairy leaves and tubular yellow flowers, surrounded by white ligulets.
DISTRIBUTION: Native to north west Africa and southern Spain, having probably evolved from the very common Ormenis species which grows all around the Mediterranean. Also found growing on the plains in Israel. The oil is distilled in Morocco.
OTHER SPECIES: It is distantly related to the German and Roman chamomile botanically, although it does not resemble them physically.
HERBAL/FOLK TRADITION: This is one of the more recent oils to appear on the market, and as such it does not have a long history of usage. The oil is often mistaken for a ‘true’ chamomile, though it should more correctly be called ‘Ormenis oil’ since: ‘Chemically and olfactorily, the oil is distinctly different from the German or the Roman chamomile oils, and cannot be considered as a replacement for them.’.
ACTIONS: Antispasmodic, cholagogue, emmenagogue, hepatic, sedative.
EXTRACTION: Essential oil by steam distillation from the flowering tops.
CHARACTERISTICS: Pale yellow to brownish yellow mobile liquid with a fresh-herbaceous top note and a sweet rich-balsamic undertone. It blends well with cypress, lavender, lavandin, vetiver, cedarwood, oakmoss, labdanum, olibanum and artemisia oils.
PRINCIPAL CONSTITUENTS: Unknown.
SAFETY DATA: Generally non-toxic and non-irritant – more specific safety data is unavailable at present.
AROMATHERAPY/HOME: USE ‘Sensitive skin, colic, colitis, headache, insomnia, irritability, migraine, amenorrhoea, dysmenorrhoea, menopause, liver and spleen congestion.’. Little is known about its therapeutic history and usage.
OTHER USES: Employed extensively in perfumery work, especially in colognes, chypres and fougère fragrance.... chamomile, maroc
FAMILY: Rutaceae
SYNONYMS: C. racemosa, C. maxima var. racemosa, shaddock (oil).
GENERAL DESCRIPTION: A cultivated tree, often over 10 metres high with glossy leaves and large yellow fruits, believed to have derived from the shaddock (C. grandis).
DISTRIBUTION: Native to tropical Asia, and the West Indies; cultivated in California, Florida, Brazil and Israel. The oil is mainly produced in California.
OTHER SPECIES: C. paradisi is a recent hybrid of C. maxima and C. sinesis. There are many different cultivars; for example, ‘Duncan’ is standard in Florida.
HERBAL/FOLK TRADITION: It shares the nutritional qualities of other citrus species, being high in Vitamin C and a valuable protection against infectious illness.
ACTIONS: Antiseptic, antitoxic, astringent, bactericidal, diuretic, depurative, stimulant (lymphatic, digestive), tonic.
EXTRACTION: Essential oil by cold expression from the fresh peel. (Some oil is distilled from the peel and remains of the fruit after the juice has been utilized, but this is of inferior quality.)
CHARACTERISTICS: A yellow or greenish mobile liquid with a fresh, sweet citrus aroma. It blends well with lemon, palmarosa, bergamot, neroli, rosemary, cypress, lavender, geranium, cardomon and other spice oils.
PRINCIPAL CONSTITUENTS: Limonene (90 per cent), cadinene, paradisiol, neral, geraniol, citronellal, sinensal, as well as esters, coumarins and furocoumarins.
SAFETY DATA: Non-toxic, non-irritant, non-sensitizing, non-phototoxic. It has a short shelf life – it oxidizes quickly.
AROMATHERAPY/HOME: USE
SKIN CARE: Acne, congested and oily skin, promotes hair growth, tones the skin and tissues.
Circulation Muscles And Joints: Cellulitis, exercise preparation, muscle fatigue, obesity, stiffness, water retention.
Immune System: Chills, colds, ’flu.
Nervous System: Depression, headaches, nervous exhaustion, performance stress.
OTHER USES: Employed as a fragrance component in soaps, detergents, cosmetics and perfumes. Extensively used in desserts, soft drinks and alcoholic beverages.... grapefruit
FAMILY: Lauraceae
SYNONYMS: Sweet bay, bay laurel, Grecian laurel, true bay, Mediterranean bay, Roman laurel, noble laurel, laurel leaf (oil).
GENERAL DESCRIPTION: An evergreen tree up to 20 metres high with dark green, glossy leaves and black berries; often cultivated as an ornamental shrub.
DISTRIBUTION: Native to the Mediterranean region; extensively cultivated especially for its berries, in France, Spain, Italy, Morocco, Yugoslavia, China, Israel, Turkey and Russia. The oil is mainly produced in Yugoslavia.
OTHER SPECIES: There are several related species, all of which are commonly called Bay: Californian bay (Umbellularia california), West Indian bay (Pimenta racemosa) and the cherry laurel (Prunus laurocerasus), which is poisonous.
HERBAL/FOLK TRADITION: A popular culinary herb throughout Europe. The leaves were used by the ancient Greeks and Romans to crown their victors. Both leaf and berry were formerly used for a variety of afflictions including hysteria, colic, indigestion, loss of appetite, to promote menstruation and for fever. It is little used internally these days, due to its narcotic properties. A ‘fixed’ oil of bay, expressed from the berries, is still used for sprains, bruises, earache, etc.
ACTIONS: Antirheumatic, antiseptic, bactericidal, diaphoretic, digestive, diuretic, emmenagogue, fungicidal, hypotensive, sedative, stomachic.
EXTRACTION: Essential oil by steam distillation from the dried leaf and branchlets. (An oil from the berries is produced in small quantities.)
CHARACTERISTICS: A greenish-yellow liquid with a powerful, spicy-medicinal odour. It blends well with pine, cypress, juniper, clary sage, rosemary, olibanum, labdanum, lavender, citrus and spice oils.
PRINCIPAL CONSTITUENTS: Cineol (30–50 per cent), pinene, linalol, terpineol acetate, and traces of methyl eugenol.
SAFETY DATA: Relatively non-toxic and nonirritant; can cause dermatitis in some individuals. Use in moderation due to possible narcotic properties attributed to methyl eugenol. Should not be used during pregnancy.
AROMATHERAPY/HOME: USE
Digestive system: Dyspepsia, flatulence, loss of appetite.
Genito-urinary system: Scanty periods. Immune system: Colds, ’flu, tonsillitis and viral infections.
OTHER USES: Used as a fragrance component in detergents, cosmetics, toiletries and perfumes, especially aftershaves. Extensively used in processed food of all types, as well as alcoholic and soft drinks.... laurel
FAMILY: Rutaceae
SYNONYMS: C. limonum, cedro oil.
GENERAL DESCRIPTION: A small evergreen tree up to 6 metres high with serrated oval leaves, stiff thorns and very fragrant flowers. The fruit turns from green to yellow on ripening.
DISTRIBUTION: Native to Asia, probably east India; it now grows wild in the Mediterranean region especially in Spain and Portugal. It is cultivated extensively worldwide in Italy, Sicily, Cyprus, Guinea, Israel, South and North America (California and Florida).
OTHER SPECIES: There are about forty-seven varieties which are said to have been developed in cultivation, such as the Java lemon (C. javanica). The lemon is also closely related to the lime, cedrat (or citron) and bergamot.
HERBAL/FOLK TRADITION: The juice and peel are widely used as a domestic seasoning. It is very nutritious, being high in vitamins A, B and C. In Spain and other European countries, lemon is something of a ‘cure-all’, especially with regard to infectious illness. It was used for fever, such as malaria and typhoid, and employed specifically for scurvy on English ships at sea.
Taken internally, the juice is considered invaluable for acidic disorders, such as arthritis and rheumatism, and of great benefit in dysentery and liver congestion.
ACTIONS: Anti-anaemic, antimicrobial, antirheumatic, antisclerotic, antiscorbutic, antiseptic, antispasmodic, antitoxic, astringent, bactericidal, carminative, cicatrisant, depurative, diaphoretic, diuretic, febrifuge, haemostatic, hypotensive, insecticidal, rubefacient, stimulates white corpuscles, tonic, vermifuge.
EXTRACTION: Essential oil by cold expression from the outer part of the fresh peel. A terpeneless oil is also produced on a large scale (cedro oil).
CHARACTERISTICS: A pale greeny-yellow liquid (turning brown with age), with a light, fresh, citrus scent. It blends well with lavender, neroli, ylang ylang, rose, sandalwood, olibanum, chamomile, benzoin, fennel, geranium, eucalyptus, juniper, oakmoss, lavandin, elemi, labdanum and other citrus oils.
PRINCIPAL CONSTITUENTS: Limonene (approx. 70 per cent), terpinene, pinenes, sabinene, myrcene, citral, linalol, geraniol, octanol, nonanol, citronellal, bergamotene, among others.
SAFETY DATA: Non-toxic; may cause dermal irritation or sensitization reactions in some individuals – apply in moderation. Phototoxic do not use on skin exposed to direct sunlight.
AROMATHERAPY/HOME: USE
Skin care: Acne, anaemia, brittle nails, boils, chilblains, corns, cuts, greasy skin, herpes, insect bites, mouth ulcers, spots, varicose veins, warts.
Circulation muscles and joints: Arthritis, cellulitis, high blood pressure, nosebleeds, obesity (congestion), poor circulation, rheumatism.
Respiratory system: Asthma, throat infections, bronchitis, catarrh.
Digestive system: Dyspepsia.
Immune system: Colds, ’flu, fever and infections.
OTHER USES: Used as a flavouring agent in pharmaceuticals. Extensively used as a fragrance component in soaps, detergents, cosmetics, toilet waters and perfumes. Extensively employed by the food industry in most types of product, including alcoholic and soft drinks.... lemon
FAMILY: Rutaceae
SYNONYMS: C. vulgaris, C. bigaradia, Seville orange, sour orange bigarade (oil).
GENERAL DESCRIPTION: An evergreen tree up to 10 metres high with dark green, glossy, oval leaves, paler beneath, with long but not very sharp spines. It has a smooth greyish trunk and branches, and very fragrant white flowers. The fruits are smaller and darker than the sweet orange. It is well known for its resistance to disease and is often used as root stock for other citrus trees, including the sweet orange.
DISTRIBUTION: Native to the Far East, especially India and China, but has become well adapted to the Mediterranean climate. It also grows abundantly in the USA (California), Israel and South America. Main producers of the oil include Spain, Guinea, the West Indies, Italy, Brazil and the USA.
OTHER SPECIES: There are numerous different species according to location – oils from Spain and Guinea are said to be of superior quality.
HERBAL/FOLK TRADITION: ‘Oranges and lemons strengthen the heart, are good for diminishing the coagubility of the blood, and are beneficial for palpitation, scurvy, jaundice, bleedings, heartburn, relaxed throat, etc. They are powerfully anti-scorbutic, either internally or externally applied.’. The dried bitter orange peel is used as a tonic and carminative in treating dyspepsia.
In Chinese medicine the dried bitter orange and occasionally its peel are used in treating prolapse of the uterus and of the anus, diarrhoea, and blood in the faeces. Ingestion of large amounts of orange peel in children, however, has been reported to cause toxic effects.
ACTIONS: Anti-inflammatory, antiseptic, astringent, bactericidal, carminative, choleretic, fungicidal, sedative (mild), stomachic, tonic.
EXTRACTION: An essential oil by cold expression (hand or machine pressing) from the outer peel of the almost ripe fruit. (A terpeneless oil is also produced.) The leaves are used for the production of petitgrain oil; the blossom for neroli oil.
CHARACTERISTICS: A dark yellow or brownish-yellow mobile liquid with a fresh, dry, almost floral odour with a rich, sweet undertone.
PRINCIPAL CONSTITUENTS: Over 90 per cent monoterpenes: mainly limonene, myrcene, camphene, pinene, ocimene, cymene, and small amounts of alcohols, aldehydes and ketones.
SAFETY DATA: Phototoxic; otherwise generally non-toxic, non-irritant and non sensitizing. Limonene has been reported to cause contact dermatitis in some individuals.
AROMATHERAPY/HOME: USE See sweet orange.
OTHER USES: Used in certain stomachic, laxative and carminative preparations. Employed as a fragrance component in soaps, detergents, cosmetics, colognes and perfumes. Extensively used as a flavouring material, especially in liqueurs and soft drinks. Also utilized as a starting material for the isolation of naturallimonene.... orange, bitter
FAMILY: Rutaceae
SYNONYMS: C. aurantium var. dulcis, C. aurantium var. sinensis, China orange, Portugal orange.
GENERAL DESCRIPTION: An evergreen tree, smaller than the bitter variety, less hardy with fewer or no spines. The fruit has a sweet pulp and non-bitter membranes. Another distinguishing feature is the shape of the leaf stalk: the bitter orange is broader and in the shape of a heart.
DISTRIBUTION: Native to China; extensively cultivated especially in America (California and Florida) and round the Mediterranean (France, Spain, Italy). The expressed oil is mainly produced in Israel, Cyprus, Brazil and North America; the distilled oil mainly comes from the Mediterranean and North America.
OTHER SPECIES: There are numerous cultivated varieties of sweet orange, for example Jaffa, Navel and Valencia. There are also many other subspecies such as the Japanese orange (C. aurantium var. natsudaidai). See also bitter orange.
HERBAL/FOLK TRADITION: A very nutritious fruit, containing vitamins A, B and C. In Chinese medicine the dried sweet orange peel is used to treat coughs, colds, anorexia and malignant breast sores. Li Shih-chen says: ‘The fruits of all the different species and varieties of citrus are considered by the Chinese to be cooling. If eaten in excess they are thought to increase the “phlegm”, and this is probably not advantageous to the health. The sweet varieties increase bronchial secretion, and the sour promote expectoration. They all quench thirst, and are stomachic and carminative.’.
ACTIONS: Antidepressant, anti-inflammatory, antiseptic, bactericidal, carminative, choleretic, digestive, fungicidal, hypotensive, sedative (nervous), stimulant (digestive and lymphatic), stomachic, tonic.
EXTRACTION: 1. Essential oil by cold expression (hand or machine) of the fresh ripe or almost ripe outer peel. 2. Essential oil by steam distillation of the fresh ripe or almost ripe outer peel. An oil of inferior quality is also produced by distillation from the essences recovered as a byproduct of orange juice manufacture. Distilled sweet orange oil oxidizes very quickly, and anti-oxidant agents are often added at the place of production. (An oil from the flowers is also produced occasionally called neroli Portugal or neroli petalae; an oil from the leaves is also produced in small quantities.)
SYNONYM: 1. A yellowy-orange or dark orange mobile liquid with a sweet, fresh fruity scent, richer than the distilled oil. It blends well with lavender, neroli, lemon, clary sage, myrrh and spice oils such as nutmeg, cinnamon and clove.
2. A pale yellow or colourless mobile liquid with a sweet, light-fruity scent, but little tenacity.
PRINCIPAL CONSTITUENTS: Over 90 per cent monoterpenes, mainly limonene. The cold expressed oil also contains bergapten, auraptenol and acids.
SAFETY DATA: Generally non-toxic (although ingestion of large amounts of orange peel has been known to be fatal to children); non-irritant and non-sensitizing (although limonene has been found to cause dermatitis in a few individuals). Distilled orange oil is phototoxic: its use on the skin should be avoided if there is danger of exposure to direct sunlight. However, there is no evidence to show that expressed sweet orange oil is phototoxic although it too contains coumarins.
AROMATHERAPY/HOME: USE
Skin care: Dull and oily complexions, mouth ulcers.
Circulation muscles and joints: Obesity, palpitations, water retention.
Respiratory system: Bronchitis, chills.
Digestive system: Constipation, dyspepsia, spasm.
Immune system: Colds, ’flu.
Nervous system: Nervous tension and stressrelated conditions.
OTHER USES: Sweet orange peel tincture is used to flavour pharmaceuticals. Extensively used as a fragrance component in soaps, detergents, cosmetics and perfumes, especially eau-de-colognes. Extensively used in all areas of the food and drinks industry (more so than the bitter orange oil). Used as the starting material for the isolation of naturallimonene.... orange, sweet
FAMILY: Lamiaceae (Labiatae)
SYNONYMS: T. capitans, Coridothymus capitatus, Satureja capitata, Thymbra capitata, oreganum (oil), Israeli oreganum (oil), Cretan thyme, corido thyme, conehead thyme, headed savory, thyme of the ancients.
GENERAL DESCRIPTION: A perennial creeping herb with a woody stem, small dark green leaves and pink or white flowers borne in clusters.
DISTRIBUTION: Native to the Middle East and Asia Minor; grows wild in Spain. The oil is produced mainly in Spain, Israel, Lebanon, Syria and Turkey.
OTHER SPECIES: Although this herb is strictly a thyme, it serves as the source for most so-called ‘oregano oil’. For other related species see entries on common thyme, common oregano and sweet marjoram; see also Botanical Classification section.
HERBAL/FOLK TRADITION: According to Mrs Grieve the properties and oil of Spanish oregano (Thymus capitatus) are similar to the common thyme (T. vulgaris); it also shares many qualities with the common oregano or wild marjoram (Origanum vulgare).
ACTIONS: See common oregano.
EXTRACTION: Essential oil by steam distillation from the dried flowering tops.
CHARACTERISTICS: A dark brownish-red or purple oil with a strong tarlike, herbaceous, refreshing odour.
PRINCIPAL CONSTITUENTS: Carvacrol, thymol, cymene, caryophyllene, pinene, limonene, linalol, borneol, myrcene, thujone, terpinene.
SAFETY DATA: Dermal toxin, skin irritant, mucous membrane irritant.
AROMATHERAPY/HOME: USE None. ‘Should not be used on the skin at all.’.
OTHER USES: See common oregano.... oregano, spanish
FAMILY: Lamiaceae (Labiatae)
SYNONYMS: T. aestivus, T. ilerdensis, T. webbianus, T. valentianus, French thyme, garden thyme, red thyme (oil), white thyme (oil).
GENERAL DESCRIPTION: A perennial evergreen subshrub up to 45 cms high with a woody root and much-branched upright stem. It has small, grey-green, oval, aromatic leaves and pale purple or white flowers.
DISTRIBUTION: Native to Spain and the Mediterranean region; now found throughout Asia Minor, Algeria, Turkey, Tunisia, Israel, the USA, Russia, China and central Europe. The oil is mainly produced in Spain but also in France, Israel, Greece, Morocco, Algeria, Germany and the USA.
OTHER SPECIES: There are numerous varieties of thyme – the common thyme is believed to have derived from the wild thyme or mother-of-thyme (T. serpyllum), which is also used to produce an essential oil called serpolet, similar in effect to the common thyme oil.
Another species used for the production of the so-called red thyme oil is particularly the Spanish sauce thyme (T. zygis), a highly penetrating oil good for cellulitis, sports injuries, etc. (although, like the common thyme, it is a skin irritant). Other species used for essential oil production include lemon thyme (T. citriodorus), a fresh scented oil good for asthma and other respiratory conditions, safe for children. Spanish marjoram (T. Mastichina) is actually botanically classified as a variety of thyme, with which it shares many common properties, despite its common name. Spanish marjoram contains mainly 1,8-cineole (up to 75 per cent) and linalool (up to 20 per cent), so therapeutically has much in common with eucalyptus, being well suited to treating viral and bacterial respiratory infections. See also Botanical Classification section.
HERBAL/FOLK TRADITION: One of the earliest medicinal plants employed throughout the Mediterranean region, well known to both Hippocrates and Dioscorides. It was used by the ancient Egyptians in the embalming process, and by the ancient Greeks to fumigate against infectious illness; the name derives from the Greek thymos meaning ‘to perfume’. It is also a long-established culinary herb, especially used for the preservation of meat.
It has a wide range of uses, though in Western herbal medicine its main areas of application are respiratory problems, digestive complaints and the prevention and treatment of infection. In the British Herbal Pharmacopoeia it is indicated for dyspepsia, chronic gastritis, bronchitis, pertussis, asthma, children’s diarrhoea, laryngitis, tonsillitis and enuresis in children.
ACTIONS: Anthelmintic, antimicrobial, antioxidant, antiputrescent, antirheumatic, antiseptic (intestinal, pulmonary, genito-urinary), antispasmodic, antitussive, antitoxic, aperitif, astringent, aphrodisiac, bactericidal, balsamic, carminative, cicatrisant, diuretic, emmenagogue, expectorant, fungicidal, hypertensive, nervine, revulsive, rubefacient, parasiticide, stimulant (immune system, circulation), sudorific, tonic, vermifuge.
EXTRACTION: Essential oil by water or steam distillation from the fresh or partially dried leaves and flowering tops. 1. ‘Red thyme oil’ is the crude distillate. 2. ‘White thyme oil’ is produced by further redistillation or rectification. (An absolute is also produced in France by solvent extraction for perfumery use.)
CHARACTERISTICS: 1. A red, brown or orange liquid with a warm, spicy-herbaceous, powerful odour. 2. A clear, pale yellow liquid with a sweet, green-fresh, milder scent. It blends well with bergamot, lemon, rosemary, melissa, lavender, lavandin, marjoram, Peru balsam, pine, etc.
PRINCIPAL CONSTITUENTS: Thymol and carvacrol (up to 60 per cent), cymene, terpinene, camphene, borneol, linalol; depending on the source it can also contain geraniol, citral and thuyanol, etc.
There are many chemotypes of thyme oil: notably the ‘thymol’ and ‘carvacrol’ types (warming and active); the ‘thuyanol’ type (penetrating and antiviral); and the milder ‘linalol’ or ‘citral’ types (sweet-scented, non-irritant).
SAFETY DATA: Red thyme oil, serpolet (from wild thyme), ‘thymol’ and ‘carvacrol’ type oils all contain quite large amounts of toxic phenols (carvacrol and thymol). They can irritate mucous membranes, cause dermal irritation and may cause sensitization in some individuals. Use in moderation, in low dilution only. They are best avoided during pregnancy.
White thyme is not a ‘complete’ oil and is often adulterated. Lemon thyme and ‘linalol’types are in general less toxic, non-irritant, with less possibility of sensitization – safe for use on the skin and with children.
AROMATHERAPY/HOME: USE
Skin care: Abscess, acne, bruises, burns, cuts, dermatitis, eczema, insect bites, lice, gum infections, oily skin, scabies.
Circulation muscles and joints: Arthritis, cellulitis, gout, muscular aches and pains, obesity, oedema, poor circulation, rheumatism, sprains, sports injuries.
Respiratory system: Asthma, bronchitis, catarrh, coughs, laryngitis, sinusitis, sore throat, tonsillitis.
Digestive system: Diarrhoea, dyspepsia, flatulence.
Genito-urinary system: Cystitis, urethritis.
Immune system: Chills, colds, ’flu, infectious diseases.
Nervous system: Headaches, insomnia, nervous debility and stress-related complaints – ‘helps to revive and strengthen both body and mind’..
OTHER USES: The oil is used in mouthwashes, gargles, toothpastes and cough lozenges. ‘Thymol’ is isolated for pharmaceutical use in surgical dressings, disinfectants etc. Used as a fragrance component in soaps, toiletries, aftershaves, perfumes, colognes, etc. Extensively employed by the food and drink industry, especially in meat products.... thyme, common