A sudden attack, worsening, or recurrence of symptoms or of a disease; a spasm or seizure.
Cyclic manifestation of acute illness in malaria, characterised by a rise in temperature with accompanying symptoms, usually caused by invasion of the blood by a brood of parasites released from RBC’s.
A sudden temporary attack that may take the form of a convulsion or spasm. It may also occur when a patient with a disease suddenly deteriorates.
n. 1. a sudden violent attack, especially a spasm or convulsion. 2. the abrupt worsening of symptoms or recurrence of disease. —paroxysmal adj.
Attacks of uncontrollable coughing or “whooping”, often relating to whooping cough or bronchiectasis, but they can also be caused by the smoke from burning plastics and (memories of yesteryear) hash oil.... cough, paroxysmal
(BPPV) a common cause of vertigo in which the patient complains of brief episodes of rotatory vertigo precipitated by sudden head movements. It is thought to be due to microscopic debris derived from the *otoliths of the utricle and displaced into one of the semicircular canals, most commonly the posterior semicircular canal. The debris is most commonly thought to be free in the canal (canalithiasis; see canalith) but can be attached to the *cupula (cupulolithiasis; see cupulolith). Diagnosis is by performing a *Dix–Hallpike test. Treatment is with a predetermined set of head movements to move the debris from the semicircular canal (see Epley particle repositioning manoeuvre; Semont liberatory manoeuvre; Brandt-Daroff exercises). Surgery is occasionally used to occlude the relevant semicircular canal, cut the *singular nerve or vestibular nerves, or perform a *labyrinthectomy. Drugs are generally ineffective in the treatment of this condition.... benign paroxysmal positional vertigo
(PNH) a type of acquired haemolytic *anaemia that results from an abnormality of the red blood cell membrane. It is due to a defect in the formation of glycosylphosphatidylinositol (GPI), whose role is to anchor proteins to the lipid framework of the membrane. This leads to increased *complement-mediated destruction of red blood cells, which results in the release of haemoglobin in the circulation and then in the urine, giving the latter a reddish colour. Some patients may develop blood clots.... paroxysmal nocturnal haemoglobinuria
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